Expression of aberrant forms of CD22 on B lymphocytes in Cd22^a lupus-prone mice affects ligand binding
スポンサーリンク
概要
- 論文の詳細を見る
- 2006-01-01
著者
-
IZUI Shozo
Department of Pathology, Center Medical Universitaire, University of Geneva
-
KIKUCHI SHUICHI
Department of Neuropsychiatry, Faculty of Medicine, Chiba University
-
Kikuchi Shuichi
Department Of Neuropsychiatry Faculty Of Medicine Chiba University
-
Kikuchi Shuichi
Department Of Pathology And Immunology University Medical Center Cmu
-
NITSCHKE Lars
Department of Genetics, University of Erlangen
-
Nitschke Lars
Department Of Genetics University Of Erlangen
-
Izui Shozo
Department Of Pathology And Immunology University Medical Center Cmu
-
Parkhouse R.
Gulbenkian Institute For Science
-
LAJAUNIAS Frederic
Department of Pathology and Immunology, University Medical Center, CMU
-
MOLL Thomas
Department of Pathology and Immunology, University Medical Center, CMU
-
HO Liza
Department of Pathology and Immunology, University Medical Center, CMU
-
MARTINEZ-SORIA Eduardo
Department of Pathology and Immunology, University Medical Center, CMU
-
SANTIAGO-RABER Marie-Laure
Department of Pathology and Immunology, University Medical Center, CMU
-
DIX Carolin
Department of Genetics, University of Erlangen
-
Santiago-raber Marie-laure
Department Of Pathology And Immunology Faculty Of Medicine University Of Geneva
-
Ho Liza
Department Of Pathology And Immunology University Medical Center Cmu
-
Dix Carolin
Department Of Genetics University Of Erlangen
-
Moll Thomas
Department Of Pathology And Immunology University Medical Center Cmu
-
Lajaunias Frederic
Department Of Pathology And Immunology University Medical Center Cmu
-
Martinez-soria Eduardo
Department Of Pathology And Immunology University Medical Center Cmu
-
Santiago-raber Marie-laure
Department Of Pathology And Immunology University Medical Center Cmu
関連論文
- Suppression of experimental lupus nephritis by aberrant expression of the soluble E-selectin gene
- Molecular changes of NMDA receptor subunit mRNA in amygdala-kindled rat
- Abnormal lgG galactosylation in MRL-Ipr/Ipr mice : Pathogenic role in the development of arthritis
- Expression of aberrant forms of CD22 on B lymphocytes in Cd22^a lupus-prone mice affects ligand binding
- Regulation of B cell development and B cell signalling by CD22 and its ligands α2,6-linked sialic acids
- Immunopathogenesis of lupus nephritis:New insights from experimental models
- Tonic B cell activation by Radioprotective105/MD-1 promotes disease progression in MRL/Ipr mice
- T Helper Subsets and Corresponding IgG Subclass Responses in the Pathogenesis of Murine Lupus-Like Autoimmune Syndrome
- Glatiramer acetate treatment does not modify the clinical course of (NZB×BXSB)F1 lupus murine model