The induction mechanism of no synthase in experimental models of acute pancreatitis
スポンサーリンク
概要
- 論文の詳細を見る
- 1997-03-17
著者
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SATOH Akihiko
Third Department of Internal Medicine, Tohoku University School of Medicine
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Satoh Akihiko
Third Department Of Internal Medicine Tohoku University School Of Medicine
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SHIMOSEGAWA Toooru
Third Department of Internal Medicine, Tohoku University School of Medicine
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Shimosegawa Toooru
Third Department Of Internal Medicine Tohoku University School Of Medicine
関連論文
- Gallbladder emptying and cholecystokinin and pancreatic polypeptide responses to a liquid meal in patients with diabetes mellitus
- Ascitic Fluid of Experimental Severe Acute Pancreatitis Modulates the Function of Peritoneal Macrophages
- Nitric Oxide Is Overproduced by Peritoneal Macrophages in Rat Taurocholate Pancreatitis : The Mechanism of Inducible Nitric Oxide Synthase Expression
- Low Doses of Lipopolysaccharide Upregulate Acinar Cell Apoptosis in Cerulein Pancreatitis
- The Role of Nitric Oxide in Mouse Cerulein-Induced Pancreatitis With and Without Lipopolysaccharide Pretreatment
- Lysophosphatidylcholine Induces Apoptosis in AR42J Cells
- Nitric Oxide Decreases Endothelial Activation by Rat Experimental Severe Pancreatitis-Associated Ascitic Fluids
- Gastric mucosal blood flow response to stress in streptozotocin diabetic rats : Regulatory role of nitric oxide
- The induction mechanism of no synthase in experimental models of acute pancreatitis