Ig gene somatic hypermutation in mice defective for DNA polymerase δ proofreading
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概要
- 論文の詳細を見る
- 2003-04-01
著者
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Goldsby Robert
Department Of Pediatrics University Of California At San Francisco
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STORB Ursula
Department of Molecular Genetics and Cell Biology, University of Chicago
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LONGACRE Angelika
Department of Molecular Genetics and Cell Biology, University of Chicago
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SUN Tianhe
Department of Molecular Genetics and Cell Biology, University of Chicago
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PRESTON Bradley
Department of Pathology, University of Washington
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Storb Ursula
Department Of Molecular Genetics And Cell Biology And Biochemistry And Molecular Biology University
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Sun Tianhe
Department Of Molecular Genetics And Cell Biology University Of Chicago
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Preston Bradley
Department Of Pathology University Of Washington
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Longacre Angelika
Department Of Molecular Genetics And Cell Biology University Of Chicago
関連論文
- γ2b provides only some of the signals normally given via μ in B cell development
- Ig gene somatic hypermutation in mice defective for DNA polymerase δ proofreading
- The TATA binding protein, c-Myc and survivin genes are not somatically hypermutated, while lg and BCL6 genes are hypermutated in human memory B cells
- The 3' lgκ enhancer contains RNA polymerase II promoters : implications for endogenous and transgenic κ gene expression
- The C_H 1 and transmembrane domains of u in the context of a γ2b transgene do not suffice to promote B cell maturation