Receptors and Ligands for Autocrine Growth Pathways Are Up-regulated When Pancreatic Cancer Cells Are Adapted to Serum-Free Culture
スポンサーリンク
概要
- 論文の詳細を見る
- 2001-04-01
著者
-
Adrian Thomas
Department Of Biomedical Sciences Creighton University School Of Medicine
-
Murphy Leon
Department Of Biomedical Sciences Creighton University School Of Medicine
-
Adrian Thomas
Department Of Biomedical Sciences And Cancer Center Creighton University School Of Medicine
-
ABDEL-WAHAB Yasser
Department of Biomedical Sciences, Creighton University School of Medicine
-
WANG Qiming
Department of Biomedical Sciences, Creighton University School of Medicine
-
KNEZETIC Joseph
Department of Biomedical Sciences, Creighton University School of Medicine
-
PERMERT Johan
Department of Surgery, Karolinska Institute, Huddinge Hospital
-
LARSSON Jorgen
Department of Surgery, Karolinska Institute, Huddinge Hospital
-
HOLLINGSWORTH Anthony
Eppley Institute for Research in Cancer, University of Nebraska Medical Center
-
Permert Johan
Department Of Surgery Karolinska Institute Huddinge Hospital
-
Permert Johan
Department Of Surgery Karolinska Institute At Huddinge University Hospital
-
Abdel-wahab Yasser
Department Of Biomedical Sciences Creighton University School Of Medicine
-
Knezetic Joseph
Department Of Biomedical Sciences Creighton University School Of Medicine
-
Wang Qiming
Department Of Biomedical Sciences Creighton University School Of Medicine
-
Larsson Jorgen
Department Of Surgery Karolinska Institute Huddinge Hospital
-
Hollingsworth Anthony
Eppley Institute For Research In Cancer University Of Nebraska Medical Center
関連論文
- Receptors and Ligands for Autocrine Growth Pathways Are Up-regulated When Pancreatic Cancer Cells Are Adapted to Serum-Free Culture
- Physiological Concentrations of Insulin Augment Pancreatic Cancer Cell Proliferation and Glucose Utilization By Activating MAP Kinase, PI_3 Kinase and Enhancing GLUT-1 Expression
- On the Importance of Cholecystokinin in Neonatal Pancreatic Growth and Secretory Development in Guinea Pigs
- Transdifferentiation of Human Islet Cells in a Long-term Culture
- Establishment of Human Pancreatic Ductal Cells in a Long-Term Culture
- Islet Amyloid Polypeptide Regulates Multiple Steps in Stimulus-Secretion Coupling of β Cells in Rat Pancreatic Islets
- The Role of Oxygen-Derived Free Radicals and Nitric Oxide in Cytokine-Induced Antiproliferation of Pancreatic Cancer Cells
- Dissociated Insulin and Islet Amyloid Polypeptide Secretion from Isolated Rat Pancreatic Islets Cocultured with Human Pancreatic Adenocarcinoma Cells
- Differential Inhibition of Insulin and Islet Amyloid Polypeptide Secretion by Intraislet Somatostatin in the Isolated Perfused Human Pancreas
- The Regulation of Amylin and Insulin Gene Expression and Secretion
- Arsenic Trioxide Induces Apoptosis in Pancreatic Cancer Cells via Changes in Cell Cycle, Caspase Activation, and GADD Expression
- The Intracellular Mechanism of Insulin Resistance in the Hamster Pancreatic Ductal Adenocarcinoma Model