Conditional gene silencing utilizing the lac repressor reveals a role of SHP-2 in cagA-positive Helicobacter pylori pathogenicity
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概要
- 論文の詳細を見る
- 2004-05-10
著者
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Hatakeyama M
Hokkaido Univ. Sapporo Jpn
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Hatakeyama Masanori
Division Of Molecular Oncology Institute For Genetic Medicine Hokkaido University
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Hatakeyama Masanori
Division Of Molecular Oncology Institute For Genetic Medicine And Division Of Chemistry Graduate Sch
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HIGASHI Hideaki
Division of Molecular Oncology, Institute for Genetic Medicine, and Division of Chemistry, Graduate
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Higashi Hideaki
Division Of Molecular Oncology Institute For Genetic Medicine And Division Of Chemistry Graduate Sch
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HIGUCHI Megumi
Division of Molecular Oncology, Institute for Genetic Medicine, Hokkaido University
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TSUTSUMI Ryouhei
Division of Molecular Oncology, Institute for Genetic Medicine, Hokkaido University
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Higuchi Megumi
Division Of Molecular Oncology Institute For Genetic Medicine Hokkaido University
関連論文
- Structural and functional diversity in the PAR1b/MARK2-binding region of Helicobacter pylori CagA
- Influence of EPIYA-repeat polymorphism on the phosphorylation-dependent biological activity of Helicobacter pylori CagA
- Focal Adhesion Kinase Is a Substrate and Downstream Effector of SHP-2 Complexed with Helicobacter pylori CagA
- Conditional gene silencing utilizing the lac repressor reveals a role of SHP-2 in cagA-positive Helicobacter pylori pathogenicity
- O-ADP-Ribosylation in the NAD/NADase System : 2-Alkanols as Efficient Substrates
- Helicobacter pylori CagA : a new paradigm for bacterial carcinogenesis
- Deregulation of SHP-2 tyrosine phosphatase by the Helicobacter pylori virulence factor CagA
- Helicobacter pylori and gastric carcinogenesis
- Unusual Enzymatic Hydrolysis of NAD by Solubilized Form of NAD^+ Glycohydrolase