Immune modulation with high-dose heat-shock protein gp96 : therapy of murine autoimmune diabetes and encephalomyelitis
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概要
- 論文の詳細を見る
- 2004-04-01
著者
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Srivastava Pramod
Center For Immunotherapy Of Cancer And Infectious Diseases University Of Connecticut School Of Medic
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Srivastava Pramod
Center For Immunotherapy Of Cancer And Infectious Diseases University Of Connecticut School Of Medic
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CHANDAWARKAR Rajiv
Center for Immunotherapy of Cancer and Infectious Diseases, University of Connecticut School of Medi
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WAGH Mihir
Center for Immunotherapy of Cancer and Infectious Diseases, University of Connecticut School of Medi
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KOVALCHIN Joseph
Center for Immunotherapy of Cancer and Infectious Diseases, University of Connecticut School of Medi
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Wagh Mihir
Center For Immunotherapy University Of Connecticut School Of Medicine
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Kovalchin Joseph
Center For Immunotherapy University Of Connecticut School Of Medicine
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Chandawarkar Rajiv
Center For Immunotherapy University Of Connecticut School Of Medicine
関連論文
- Necrotic but not apoptotic cell death releases heat shock proteins, which deliver a partial maturation signal to dendritic cells and activate the NF-kB pathway
- HEAT SHOCK PROTEINS IN CANCER IMMUNOTHERAPY
- Fever-like temperature induces maturation of dendritic cells through induction of hsp90
- Immune modulation with high-dose heat-shock protein gp96 : therapy of murine autoimmune diabetes and encephalomyelitis
- In vivo delivery of heat shock protein 70 accelerates wound healing by up-regulating macrophage-mediated phagocytosis