Role of L-selectin in the development of autoimmune diabetes in non-obese diabetic mice
スポンサーリンク
概要
- 論文の詳細を見る
- 2004-02-01
著者
-
FLAVELL Richard
Section of Immunobiology, Yale University School of Medicine
-
Flavell Richard
Section Of Immunobiology Yale University School Of Medicine
-
Grewal Iqbal
Section Of Immunobiology Yale University School Of Medicine
-
Mora Conchi
Section Of Immunobiology Yale University School Of Medicine
-
WONG F.
Section of Immunobiology, Yale University School of Medicine
-
Wong F.
Section Of Immunobiology Yale University School Of Medicine
関連論文
- Roles of caspase-1 in Listeria infection in mice
- Activation of NF-κB promotes the transition of large, CD43^+ pre-B cells to small, CD43^- pre-B cells
- IL9 leads to airway inflammation by inducing IL13 expression in airway epithelial cells
- Induction of experimental autoimmune encephalomyelitis in the absence of c-Jun N-terminal kinase 2
- CIITA activates the expression of MHC class II genes in mouse T cells
- CD154-CD40-independent up-regulation of B7-2 on splenic antigen-presenting cells and efficient T cell priming by staphylococcal enterotoxin A
- Role of L-selectin in the development of autoimmune diabetes in non-obese diabetic mice
- A crucial role of CD4 T cells as a functional source of CD154 in the initiation of insulin-dependent diabetes mellitus in the non-obese diabetic mouse
- Transgenic mice which overproduce Th2 cytokines develop spontaneous atopic dermatitis and asthma
- TGF-β signaling regulates CD8+ T cell responses to high- and low-affinity TCR interactions