Inhibited proliferation of fibroblasts derived from chronic diabetic wounds and normal dermal fibroblasts treated with high glucose is associated with increased formation of L-lactate
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概要
- 論文の詳細を見る
- 1998-03-01
著者
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Hansson Anders
The Department Of Molecular Medicine The Endocrine And Diabetes Unit Karolinska Institute And Hospit
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HEILBORN Johan
Department of Dermatology and Venereology, Karolinska Institute and Hospital
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HEHENBERGER Karin
Department of Molecular Medicine, The Endocrine and Diabetes Unit, Karolinska Institute and Hospital
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BRISMAR Kerstin
Department of Molecular Medicine, The Endocrine and Diabetes Unit, Karolinska Institute and Hospital
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HANSSON Anders
Department of Molecular Medicine, The Endocrine and Diabetes Unit, Karolinska Institute and Hospital
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Heilborn Johan
Department Of Dermatology And Venereology Karolinska Institute And Hospital
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Brismar Kerstin
The Department Of Molecular Medicine The Endocrine And Diabetes Unit Karolinska Institute And Hospit
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Brismar Kerstin
Department Of Endocrinology And Diabetology Karolinska Institute And Hospital
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Hehenberger Karin
The Department Of Molecular Medicine The Endocrine And Diabetes Unit Karolinska Institute And Hospit
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Hehenberger Karin
Department Of Molecular Medicine The Endocrine And Diabetes Unit Karolinska Institute And Hospital
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Hehenberger Karin
The Department Of Molecular Medicine The Endocrine And Diabetes Unit Karolinska Institute And Hospit
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Hehenberger Karin
Department Of Endocrinology And Diabetology Karolinska Institute And Hospital
関連論文
- Impaired proliferation and increased L-lactate production of dermal fibroblasts in the GK-rat, a spontaneous model of non-insulin dependent diabetes mellitus
- Inhibited proliferation of fibroblasts derived from chronic diabetic wounds and normal dermal fibroblasts treated with high glucose is associated with increased formation of L-lactate
- Fibroblasts derived from human chronic diabetic wounds have a decreased proliferation rate, which is recovered by the addition of heparin
- Dose-dependent hyperbaric oxygen stimulation of human fibroblast proliferation
- Langerhans cells in prurigo nodularis investigated by HLA-DR and S-100 immunofluorescence double staining