The PU.1 and NF-EM5 binding motifs in the lgκ 3´ enhancer are responsible for directing somatic hypermutations to the intrinsic hotspots in the transgenic V_κ gene
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概要
- 論文の詳細を見る
- 2001-11-01
著者
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TAKEMORI Toshitada
Department of Immunology, National Institute of Infectious Diseases
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Takemori T
Department Of Immunology National Institute Of Infectious Diseases
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Takemori Toshitada
Department Of Immunology Japnese Institute Of Infectious Diseases
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Kodama Masami
Department Of Biophysics And Biochemistry Graduate School Of Science University Of Tokyo
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Hayashi Reiko
Department Of Biophysics And Biochemistry Graduate School Of Science University Of Tokyo
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Hayashi Reiko
Department Of Anesthesiology And Critical Care Division Of Clinical Medical Science Graduate School
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Sakano Hitoshi
Department Of Biophysics And Biochemistry Graduate School Of Science University Of Tokyo
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Nishizumi H
Department Of Biophysics And Biochemistry Graduate School Of Science University Of Tokyo
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Nishizumi Hirofumi
Department Of Biophysics And Biochemistry Graduate School Of Science The University Of Tokyo
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Nagawa Fumikiyo
Department Of Biophysics And Biochemistry Graduate School Of Science University Of Tokyo
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- The DNA-bending protein, HMG1, is required for correct cleavage of 23 bp recombination signal sequences by recombination activating gene proteins in vitro
- The PU.1 and NF-EM5 binding motifs in the lgκ 3´ enhancer are responsible for directing somatic hypermutations to the intrinsic hotspots in the transgenic V_κ gene