Alcohol-induced sinusoidal endothelial cell dysfunction in the mouse is associated with exacerbated liver apoptosis and can be reversed by caspase inhibition
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概要
- 論文の詳細を見る
- 2001-01-01
著者
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D'souza Nympha
Division Of Pulmonary/critical Care Department Of Internal Medicine University Of Kentucky College O
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Hill Daniell
Division Of Gastroenterology/hepatology Department Of Internal Medicine University Of Louisville
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DEACIUC Ion
Division of Digestive Diseases and Nutrition, Department of Iinternal Medicine, University of Kentuc
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SARPHIE Theodore
Department of Anatomy and Cell Biology, Louisiana State University Health Sciences Center
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MCCLAIN Craig
Division of Gastroenterology/Hepatology, Department of Internal Medicine, University of Louisville
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FORTUNATO Franco
Division of Digestion Diseases and Nutrition, Department of Internal Medicine, University of Kentuck
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MCCLAIN Craig
Office of Research and Development, Medical Research Service, Department of Veterans Affairs and Uni
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Fortunato Franco
Divisions Of Digestive Diseases And Nutrition Department Of Internal Medicine University Of Kentucky
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Mcclain Craig
Division Of Gastroenterology/hepatology Department Of Internal Medicine University Of Louisville
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Deaciuc Ion
Division Of Digestive Diseases And Nutrition Department Of Iinternal Medicine University Of Kentucky
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Sarphie Theodore
Department Of Anatomy And Cell Biology Louisiana State University Health Sciences Center
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SARPHIE Theodore
Department of Anatomy and Cell Biology, Louisiana State University Medical Center
関連論文
- The regulation of Fas (CD95/Apo-1)-mediated liver apoptosis in Kupffer cell-depleted mice
- Chronic alcohol exposure of rats exacerbates apoptosis in hepatocytes and sinusoidal endothelial cells
- Alcohol-induced sinusoidal endothelial cell dysfunction in the mouse is associated with exacerbated liver apoptosis and can be reversed by caspase inhibition
- Alcohol Feeding and Lipopolysaccharide Injection Modulate Apoptotic Effectors in the Rat Pancreas In Vivo