Lactate elicits vascular endothelial growth factor from macrophages: a possible alternative to hypoxia
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概要
- 論文の詳細を見る
- 2000-09-01
著者
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Hussain M.
Department Of Surgery University Of California
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Hunt Thomas
Department Of Surgery University Of California
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Scheuenstuhl Heinz
Department Of Surgery University Of California
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CONSTANT James
the Departments of Surgery and Restorative Dentistry, University of California
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FENG John
the Departments of Surgery and Restorative Dentistry, University of California
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ZABEL David
the Departments of Surgery and Restorative Dentistry, University of California
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YUAN Hui
the Departments of Surgery and Restorative Dentistry, University of California
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SUH David
the Departments of Surgery and Restorative Dentistry, University of California
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SCHEUENSTUHL Heinz
the Departments of Surgery and Restorative Dentistry, University of California
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HUNT Thomas
the Departments of Surgery and Restorative Dentistry, University of California
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HUSSAIN M.
the Departments of Surgery and Restorative Dentistry, University of California
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CONSTANT JAMES
Wound Healing Research Laboratory, University of California
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FENG JOHN
Wound Healing Research Laboratory, University of California
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Yuan Hui
The Departments Of Surgery And Restorative Dentistry University Of California
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Zabel David
The Departments Of Surgery And Restorative Dentistry University Of California
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Feng John
The Departments Of Surgery And Restorative Dentistry University Of California
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Suh David
The Departments Of Surgery And Restorative Dentistry University Of California
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Hunt Thomas
The Department Of Surgery
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Constant James
The Departments Of Surgery And Restorative Dentistry University Of California
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Hunt Thomas
The Departments Of Surgery And Restorative Dentistry University Of California
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Scheuenstuhl Heinz
The Departments Of Surgery And Restorative Dentistry University Of California
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Scheuenstuhl Heinz
The Department Of Surgery
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Hunt Thomas
Department Of Surgery
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Hussain M.
Department Of Preventive & Restorative Dental Sciences University Of California
関連論文
- Relaxin induces vascular endothelial growth factor expression and angiogenesis selectively at wound sites
- Lactate stimulates endothelial cell migration
- Lactate and oxygen constitute a fundamental regulatory mechanism in wound healing
- Lactate elicits vascular endothelial growth factor from macrophages: a possible alternative to hypoxia
- Examintion of expanded polytetrafluoroethylene wound healing models
- Hyperoxia and angiogenesis
- Stimulation of fibroblast proliferation by lactate-mediated oxidants
- Comparison of inflammatory and systemic sources of growth factors in acute and chronic human wounds
- Age-dependency of insulin-like growth factors, insulin-like growth factor-binding proteins, and acid labile subunit in plasma and wounds of surgical patients
- Role of ADP-ribosylation in wound repair. The contributions of Thomas K. Hunt, MD
- Hyperoxia improves microvascular perfusion in a murine wound model
- Perflubron emulsion increases subcutaneous tissue oxygen tension in rats
- Effect of growth hormone replacement on wound healing in healthy older men
- Long-term results of aggressive management of diabetic foot ulcers suggest significant cost effectiveness
- Letter to the editor
- Human skin wounds : A major and snowballing threat to public health and the economy