Aberrant T cell responses to myelin antigens during clinical during clinical exacerbation in patients with multiple sclerosis
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概要
- 論文の詳細を見る
- 2001-12-01
著者
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Li Suling
Tumor Immunology Lund University
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Li S
Tumor Immunology Lund University
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Hong Jian
Department Of Neurology Baylor College Of Medicine
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Singh A.
Multiple Sclerosis Research Laboratory Baylor-methodist Multiple Sclerosis Center And Department Of
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Zhang Z.
Multiple Sclerosis Research Laboratory Baylor-methodist Multiple Sclerosis Center And Department Of
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HONG Jian
Multiple Sclerosis Research Unit, Department of Neurology and Baylor-Methodist Multiple Sclerosis Ce
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Zang C.
Multiple Sclerosis Research Laboratory Baylor-methodist Multiple Sclerosis Center And Department Of
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Killian M.
Multiple Sclerosis Research Laboratory Baylor-methodist Multiple Sclerosis Center And Department Of
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Rivera M.
Multiple Sclerosis Research Laboratory Baylor-methodist Multiple Sclerosis Center And Department Of
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TEJIDA-SIMON V.
Multiple Sclerosis Research Laboratory, Baylor-Methodist Multiple Sclerosis Center and Department of
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YANG Deye
Multiple Sclerosis Research Laboratory, Baylor-Methodist Multiple Sclerosis Center and Department of
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LI Sufang
Multiple Sclerosis Research Laboratory, Baylor-Methodist Multiple Sclerosis Center and Department of
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VAN DEN
Tissue-Typing Laboratory, Academic Hospital, University o Limburg
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Tejida-simon V.
Multiple Sclerosis Research Laboratory Baylor-methodist Multiple Sclerosis Center And Department Of
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Yang Deye
Multiple Sclerosis Research Laboratory Baylor-methodist Multiple Sclerosis Center And Department Of
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Van Den
Tissue-typing Laboratory Academic Hospital University O Limburg
関連論文
- Functional association of cytokine-induced SH2 protein and protein kinase C in activated T cells
- Distinct differences in association of MHC class I with endoplasmic reticulum proteins in wild-type, and β2-microglobulin- and TAP-deficient cell lines
- Assembly of tapasin-associated MHC class I in the absence of the transporter associated with antigen processing (TAP)
- Human anti-idiotypic T cells induced by TCR peptides corresponding to a common CDR3 sequence motif in myelin basic protein-reactive T cells
- Regulatory and pro-inflammatory phenotypes of myelin basic protein-autoreactive T cells in multiple sclerosis
- Altered expression of vasoactive intestinal peptide receptors in T lymphocytes and aberrant T_h1 immunity in multiple sclerosis
- Aberrant T cell responses to myelin antigens during clinical during clinical exacerbation in patients with multiple sclerosis
- Differential activation of signal transducer and activator of transcription (STAT)3 and STAT5 and induction of suppressors of cytokine signalling in T_h1 and T_h2 cells