Molecular and cellular basis of the altered immune response against arsonate in irradiated A/J mice autologously reconstituted
スポンサーリンク
概要
著者
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Urbain J
Univ. Libre De Bruxelles Gosselies Bel
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Mancini Isabelle
Universite De Liege Unite D'immunologie
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ISMAILI Jamila
Universite Libre de Bruxelles. Laboratory of Animal Physiology
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RAZANAJAONA Diane
Schering-Plough, Laboratory for Immunological Research
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van ACKER
Universite Libre de Bruxelles. Laboratory of Animal Physiology
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WUILMART Christian
Universite Libre de Bruxelles. Laboratory of Animal Physiology
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HEINEN Ernst
Universite de Liege, Unite d'Immunologie
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LEO Oberdan
Universite Libre de Bruxelles. Laboratory of Animal Physiology
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LEBECQUE Serge
Schering-Plough, Laboratory for Immunological Research
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URBAIN Jacques
Universite Libre de Bruxelles. Laboratory of Animal Physiology
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BRAIT Maryse
Universite Libre de Bruxelles. Laboratory of Animal Physiology
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Heinen Ernst
Universite De Liege Unite D'immunologie
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Lebecque Serge
Schering-plough Laboratory For Immunological Research
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Urbain J
Department De Biologie Moleculaire Universite Libre De Bruxelles
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Leo O
Universite Libre De Bruxelles. Laboratory Of Animal Physiology
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Razanajaona Diane
Schering-plough Laboratory For Immunological Research
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Leo Oberdan
Department Of Molecular Biology Universite Libre De Bruxelles
関連論文
- Role and regulation of IL-12 in the in vivo response to staphylococcal enterotoxin B
- The immune response induced in vivo by dendritic cells is dependent on B7-1 or B7-2, but the inhibition of both signals does not lead to tolerance
- The perinatal presence of antigen (p-azophenylarsonate) or anti-μ antibodies lead to the loss of the recurrent idiotype (CRI_A) in A/J mice
- Co-stimulation lowers the threshold for activation of naive T cells by bacterial superantigens
- Induction of T_h2 responses to soluble proteins is independent of B cell tolerance status
- The adjuvant monophosphoryl lipid A increases the function of antigen-presenting cells
- Molecular and cellular basis of the altered immune response against arsonate in irradiated A/J mice autologously reconstituted
- FcR cross-linking on monocytes results in impaired T cell stimulatory capacity