Impaired antigen presentation by murine I-A^d class II MHC molecules expressed in normal and HLA-DM-defective human B cell lines
スポンサーリンク
概要
著者
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Gautam Anand
Human Genetics Group Division Of Molecular Medicine John Curtin School Of Medical Research The Austr
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ADORINI Luciano
Roche Milano Ricerche
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Weenink Sarah
Human Genetics Group Division Of Molecular Medicine John Curtin School Of Medical Research The Austr
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MELLINS Elizabeth
Department of Pediatrics, Stanford University School of Medicine
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Mccluskey James
Center For Transfusion Medicine And Immunology Flinders Medical Center
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GUERY Jean-Charles
Roche Milano Ricerche
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AVERDUNK Holger
Human Genetics Group, Division of Molecular Medicine, John Curtin School of Medical Research, The Au
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BOSTON Tanya
Human Genetics Group, Division of Molecular Medicine, John Curtin School of Medical Research, The Au
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BOSWARVA Vicki
Center for Transfusion Medicine and Immunology, Flinders Medical Center
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Averdunk Holger
Human Genetics Group Division Of Molecular Medicine John Curtin School Of Medical Research The Austr
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Boswarva Vicki
Center For Transfusion Medicine And Immunology Flinders Medical Center
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Mellins Elizabeth
Department Of Pediatrics Stanford University School Of Medicine
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Boston Tanya
Human Genetics Group Division Of Molecular Medicine John Curtin School Of Medical Research The Austr
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Mellins Elizabeth
Department Of Paediatrics University Of Pennsylvania
関連論文
- The motif for peptide binding to the insulin-dependent diabetes mellitus-associated class II MHC molecule I-A^ validated by phage display library
- A continuous central motif of invariant chain peptides, CLIP, is essential for binding to various I-A MHC class II molecules
- Influenza A virus elevates active cathepsin B in primary murine DC
- DRα:Eβ heterrodimers in DRA transgenic mice hider expression of Eα:Eβ molecules and are more efficient in antigen presentation
- Impaired antigen presentation by murine I-A^d class II MHC molecules expressed in normal and HLA-DM-defective human B cell lines
- The mode of protein antigen administration determines preferential presentation of peptide-class II complexes by lymph node dendritic or B cells