Contribution of H-2 and non-H-2 genes in the control of mercury-induced autoimmunity
スポンサーリンク
概要
著者
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Moeller G
Department Of Immunology Wenner-gren Institute Arrhenius Laboratories For Natural Sciences Stockholm
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ABEDI-VALUGERDI Manuchehr
Department of Immunology, Wenner-Gren Instiute, Arrhenius Laboratories for Natural Sciences Stockhol
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MOLLER Goran
Department of Immunology, Wenner-Gren Instiute, Arrhenius Laboratories for Natural Sciences Stockhol
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Moller Goran
Department Of Immunology The Wenner-gren Institute Arrhenius Laboratories For Natural Sciences Stock
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Abedi-valugerdi Manuchehr
Department Of Immunology Arrhenius Laboratories For Natural Sciences Stockholm University
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Abedi-valugerdi Manuchehr
Department Of Immunology Wenner-gren Institute Arrhenius Laboratories For Natural Sciences Stockholm
関連論文
- Mercury-induced anti-nucleolar autoantobodies can transgress the membrane of living cells in vivo and in vitro
- Contribution of H-2 and non-H-2 genes in the control of mercury-induced autoimmunity
- IL-2 may be a limiting factor precluding lymphocytes from genetically resistant mice from responding to HgCl_2
- In vitro effects of HgCl_2 on murine lymphocytes. II. Selective activation of T cells expressing certain V_β TCR
- Differential sensitivity to transforming growth factor (TGF)-β of CBA and of CBA/N B cells demonstrates that the IgG2b inducing factor in synovial fluid from rheumatoid arthritis patients is not identical to TGF-β