Tumor necrosis factor receptor-1 is critically involved in the development of experimenetal autoimmune myasthenia gravis
スポンサーリンク
概要
著者
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Link H
Experimental Neurology Unit Division Of Neurology Karolinska Institutet Huddinge University Hospital
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Shi Fu-dong
Microbiology And Tumor Biology Center (mtc) Karolinska Institutet
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WANG Hua-Bing
Experimental Neurology Unit, Division of Neurology, Karolinska Institutet, Huddinge University Hospi
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LI Hulun
Experimental Neurology Unit, Division of Neurology, Karolinska Institutet, Huddinge University Hospi
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CHAMBERS Benedict
Microbiology and Tumor Biology Center (MTC), Karolinska Institutet
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LINK Hans
Experimental Neurology Unit, Division of Neurology, Karolinska Institutet, Huddinge University Hospi
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LJUGGREN Hans-Gustaf
Microbiology and Tumor Biology Center (MTC), Karolinska Institutet
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Wang Hua-bing
Experimental Neurology Unit Division Of Neurology Karolinska Institutet Huddinge University Hospital
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Ljunggren H‐g
Karolinska Inst. Stockholm Swe
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Chambers Benedict
Microbiology And Tumor Biology Center (mtc) Karolinska Institutet
関連論文
- Experimental autoimmune myasthenia gravis induction in B cell-deficient mice
- Inhibition of experimental autoimmune encephalomyelitis in Lewis rats by nasal administration of encephalitogenic MBP peptides: synergistic effects of MBP 68-86 and 87-99
- Tumor necrosis factor receptor-1 is critically involved in the development of experimenetal autoimmune myasthenia gravis