Antigem-specific cellular hyporesponsiveaess in a chronic human helminth infection is mediated by T_h3T_r1-type cytokines IL-10 and transforming growth factor-βbut not by a T_h1to T_h2 shift
スポンサーリンク
概要
著者
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Fleischer Bernhard
Bernhard-nocht-institute For Tropical Medicine
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Hoerauf A
Bernhard Nocht Institute Of Tropical Medicine
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Hoerauf Achim
Bernhard-nocht-institute Of Tropical Medicine
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DOETZE Andrea
Max Planck Institut fur Immunbiologie
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SATOGUINA Judith
Bernhard Nocht Institute of Tropical Medicine
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BURCHARD Gerd
Bernhard Nocht Institute of Tropical Medicine
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RAU Thomas
Institute of Chinical Pharmacology, University of Erlangen-Nurnberg
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LOLIGER Cornelius
University Clinic Eppendorf, University of Hamburg
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Rau Thomas
Institute Of Chinical Pharmacology University Of Erlangen-nurnberg
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Loliger Cornelius
University Clinic Eppendorf University Of Hamburg
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HOERAUF Achim
Bernhard Nocht Institute of Tropical Medicine
関連論文
- A new mechanism for IL-5-dependent helminth control: neutrophil accumulation and neutrophil-mediated worm encapsulation in murine filariasis are abolished in the absence of IL-5
- Antigem-specific cellular hyporesponsiveaess in a chronic human helminth infection is mediated by T_h3T_r1-type cytokines IL-10 and transforming growth factor-βbut not by a T_h1to T_h2 shift
- Activation-induced expression of murine CD83 on T cells and identification of a specific CD83 ligand on murine B cells
- The Xid defect imparts susceptibility to experimental murine filariosis-association with a lack of antibody and IL-10 production by B cells in response to phosphorylcholine
- Production of both IFN-γ and IL-5 by Onchocerca volvulas S1 antigen-specific CD4^+ T cells from putatively immune individuals