Ig heavy chain class switching in rag-deficient mice
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概要
著者
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ALT Frederick
Howard Hughes Medical Institute, The Children's Hospital
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Sonoda Eiichiro
Institute For Genetics University Of Cologne
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Alt Frederick
Howard Hughes Institute Harvard Medical School
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Alt Frederick
Howard Hughes Medical Institute The Children's Hospital Center For Blood Research And Departmen
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Lansford Rusty
Howard Hughes Medical Institute The Children's Hospital Center For Blood Research And Departmen
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MANIS John
Howard Hughes Medical Institute, The Children's Hospital, Center for Blood Research, and Department
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RAJEWSKY Klaus
Institute for Genetics, University of Cologne
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Rajewsky Klaus
Institute For Genetics University Of Cologne
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Manis John
Howard Hughes Medical Institute The Children's Hospital Center For Blood Research And Departmen
関連論文
- The lg heavy chain intronic enhancer core region is necessary and sufficient to promote eficient class switch recombination
- Generation of normal lymphocytes derived from N-myc-deficient embryonic stem cells
- Ig heavy chain class switching in rag-deficient mice
- Molecular genetic characterization of XRCC4 function
- The V (D) J recombination defect in the xrs-6 cell line results from a point mutation in the Ku80 gene
- Impaired B cell maturation in mice lacking Bruton's tyrosine kinase (Btk) and CD40
- An expressed neo^ro cassette provides required functions of the l_γ2b exon for class switching
- Impaired signaling via the high-affinity IgE receptor in Wiskott-Aldrich syndrome protein-deficient mast cells
- Induction of activation-induced cytidine deaminase gene expression by IL-4 and CD40 ligation is dependent on STAT6 and NFкB
- CD40 expression and function in murine B cell ontogeny
- Generating mouse models of disease through gene targeting