Continued differentiation during B lymphopoiesis requires signals in addition to cell survival
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概要
著者
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STRASSER Andreas
The Walter and Eliza Hall Institute for Medical Research, Post Office, Royal Melbourne Hospital
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Strasser Andreas
The Walter And Eliza Hall Institute For Medical Research Post Office Royal Melbourne Hospital
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TARLINTON David
The Walter and Eliza Hall Institute of Medical Research, Post Office Royal Melbourne Hospital
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CORCORAN Lynn
The Walter and Eliza Hall Institute of Medical Research, Post Office Royal Melbourne Hospital
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Corcoran Lynn
The Walter And Eliza Hall Institute Of Medical Research Po Royal Melbourne Hospital
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Corcoran Lynn
The Walter And Eliza Hall Institute Of Medical Research Post Office Royal Melbourne Hospital
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Tarlinton David
The Walter And Eliza Hall Institute Of Medical Research Post Office Royal Melbourne Hospital
関連論文
- Transgenic overexpression of human Bcl-2 in islet β cells inhibits apoptosis but dues not prevent autoimmune destruction
- Rapid B cell apoptosis induced by antigen receptor ligation does not require Fas (CD95/APO-1), the adaptor protein FADD/MORT1 or CrmA-sensitive caspases but is defective in both MRL-+/+ and MRL-lpr/lpr mice
- bcl-2 transgene expression promotes survival and reduces proliferation of CD3-CD4-CD8-T cell progenitors
- Continued differentiation during B lymphopoiesis requires signals in addition to cell survival
- Differential regulation of CD36 expression in antigen-presenting cells : Oct-2 dependence in B lymphocytes but not dendritic cells or macrophages