Induction of apoptosis by protease-defective particle preparations of human immunodeficiency virus type 1 is specific to a subset of U937-derived subclones
スポンサーリンク
概要
著者
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Luftig Ronald
Department of microbiology, Immunology and Parasitology, Louisiana State University Medical Center
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Zheng Yong-Hui
Institute of Immunological Science, Hokkaido University
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Zheng Yong-hui
Institute Of Immunological Science Hokkaido University
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Luftig Ronald
Department Of Microbiology Immunology And Parasitology Louisiana Stateuniversity Medical Center
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Kameoka Masanori
Department Of Biochemistry Nara Medical University
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KAMEOKA Masanori
Section of Serology, Institute of Immunological Science, Hokkaido University
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KIMURA Takuro
Section of Serology, Institute of Immunological Science, Hokkaido University
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Zhong Qiu
Institute of Immunological Science, Hokkaido University
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Zhong Qiu
Institute Of Immunological Science Hokkaido University
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Luftig Ronald
Department Of Microbiology Immunology And Parasitology Louisiana State University Medical Center
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Kimura Takuro
Section Of Serology Institute Of Immunologial Science Hokkaido University
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Kimura Takuro
Section Of Serology Institute Of Immunological Science Hokkaido University
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Zhong Qiu
Section Of Serology Institute Of Immunological Science Hokkaido University
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Kanda Masanori
Institute Of Immunological Science Hokkaido University
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ZHENG Yong-hui
Section of Serology, Institute of Immunologial Science, Hokkaido University
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LKUTA Kazuyoshi
Section of Serology, Institute of Immunologial Science, Hokkaido University
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Zhong Qiu
Section Of Serology Institute Of Immunologial Science Hokkaido University
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Kameoka Masanori
Section Of Serology Institute Of Immunological Science Hokkaido University
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Lkuta Kazuyoshi
Section Of Serology Institute Of Immunologial Science Hokkaido University
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Kameoka M
Section Of Serology Institute Of Immunological Science Hokkaido University
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Kameoka Masanori
Section Of Serology Institute Of Immunologial Science Hokkaido University
関連論文
- A specific T-cell subset with CD4+/CD38- markers derived from HIV-1 cariers induses apoptosis in healthy donor-derived T-lymphocytes.
- Dependence of host cell cycle for activation of human immunodeficiency virus type 1 gene expression from latency
- Superoxide generation by monocytes following infection with human cytomegalovirus.
- Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25+ killer cells in a small subset, leading to induction of apoptosis in bystander cells.
- In vivo dynamics of equine infectious anemia viruses emerging during febrile episodes: Insertions/duplications at the principal neutralizing domain.
- A clearer distinction between HIV-1 paired isolates from peripheral blood mononuclear cells of asymptomatic carriers with and without CD8+ T-cells at nef rather than env V3 loci.
- A chain section containing epitopes for cytotoxic T, B and helper T cells within a highly conserved region found in the human immunodeficiency virus type 1 Gag protein.
- Insertions, duplications and substitutions in restricted gp90 regions of equine infectious anemia virus occurred during febrile episodes in an experimentally infected horse.
- Production of doughnut-shaped, protease-defective particles from a T cell clone carrying a provirus with specific mutation in the env, pol, vpr,and nef genes.
- Interleukin-4 Up-Regulates T-Tropic Human Immunodeficiency Virus Type 1 Transcription in Primary CD4^+ CD38^+ T-Lymphocyte Subset
- Exposure of Normal Monocyte - Drived Dendritic Cells to Human Immunodeficiency Virus Type - 1 Particles Leads to the Induction of Apoptosis in Co - Cultured CD4^+ as Well as CD8^+ T Cells
- Protease-defective, gp120-containing human immunodeficiency virus type 1 particles induce apoptosis more efficiently than does wild-type virus or recombinant gp120 protein in healthy donor-derived peripheral blood T cells.
- Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25 killer cells in a small subset, leading to induction of apoptosis in bystander cells
- Viral activation from latency during retrodifferentiation of U937 cells exposed to phorbol ester followed by infection with human immunodeficiency virus type 1.
- High susceptibility of U937-derived subclone to infection with human immunodeficiency virus type 1 is correlated with virus-induced celldifferentiation and superoxide generation.
- Stimulation of human immunodeficiency virus type 1 infected cells with superoxide enhances the chemotactic motile response of CD4+ human cells: implication for virus transmission by cell-to-cell interaction.
- Maintenance of high virus load even after seroconversion in newborn cats acutely infected with feline immunodeficiency virus.
- Superinfection of a defective human immunodeficiency virus type 1 provirus-carrying T cell clone with vif or vpu mutants gives cytopathic virus particles by homologous recombination.
- Demonstration of human Borna disease virus RNA in human peripheral blood mononuclear cells.
- Extracellular Nef protein regulates productive HIV-1 infection from latency.
- Naturally occurring accessory gene mutations lead to persistent human immunodeficiency virus type 1 infection of CD4-positive T cells.
- Cytotoxic T lymphocyte response in mice induced by a recombinant BCG Vaccination which produces an extracellular α antigen that @@S ' fused with the human immunodeficiency virus type 1 envelope immuno @@S dominant domain in the V3 loop.
- Induction of apoptosis by protease-defective particle preparations of human immunodeficiency virus type 1 is specific to a subset of U937-derived subclones
- Persistently human immunodeficiency virus type 1-infected T cell CD4 expression.
- In vivo induction of human immunodeficiency virus type 1-specific cytotoxic T lymphocytes and delayed-type hypersensitivity by a @@S ' 23-amino acid peptide from the highly conserved region in major @@S ' core protein p24
- The carboxyl-terminal region of HIV-1 Nef protein is a cell surface domain that can interact with CD4+ T cells
- Molecular Ratio between Borna Disease Viral-p40 and-p24 Proteins in Infected Cells Determined by Quantitative Antigen Capture ELISA
- Archetype JC virus efficiently propagates in kidney-derived cells stably expressing HIV-1 Tat
- Multiple effects of CD4 CDR3-related peptide derivatives showing anti-HIV-1 activity on HIV-1 gp120 functions
- Superoxide enhances the spread of HIV-1 infection by cell-to-cell
- Amplification of superoxide anion generation in phagocytic cells by
- Poly(ADP-ribose) polymerase activity in various U937 cell subclones with different susceptibility to HIV-1 infection : Its dramatic decre ase following persistent virus infection.
- Alteration of Apoptotic Protease-Activating Factor-1 (APAF-1)-Dependent Apoptotic Pathway During Development of Rat Brain and Liver
- AIDS pathogenesis: the role of accessory gene mutations, leading to formation of long-lived persistently infected cells and/or apoptosis- inducing HIV-1 particles.
- Efficient propagation of progressive multifocal leukoencephalopathy-type JC virus in COS-7-derived cell lines stably expressing Tat protein of human immunodeficiency virus type 1