A model for antigen-induced T cell unresponsiveness based on autophosphorylative protein tyrosine kinase activity
スポンサーリンク
概要
著者
-
LEO Oberdan
Laboratoire de Physiologie Animale, Departement de Biologie Moleculaire, Universite Libre de Bruxell
-
Leo Oberdan
Laboratoire De Physiologie Animale Universite Libre Be Bruxelles
-
KAUFMAN Marcelle
Center for Nonlinear Phenomena and Complex Systems, Universite Libre be Bruxelles
-
ANDRIS Fabienne
Laboratoire de Physiologie Animale, Universite Libre be Bruxelles
-
Andris Fabienne
Laboratoire De Physiologie Animale Universite Libre Be Bruxelles
-
Kaufman Marcelle
Center For Nonlinear Phenomena And Complex Systems Universite Libre Be Bruxelles
-
Leo Oberdan
Laboratoire De Physiologie Animale Departement De Biologie Moleculaire Universite Libre De Bruxelles
関連論文
- Role and regulation of IL-12 in the in vivo response to staphylococcal enterotoxin B
- The immune response induced in vivo by dendritic cells is dependent on B7-1 or B7-2, but the inhibition of both signals does not lead to tolerance
- The perinatal presence of antigen (p-azophenylarsonate) or anti-μ antibodies lead to the loss of the recurrent idiotype (CRI_A) in A/J mice
- Co-stimulation lowers the threshold for activation of naive T cells by bacterial superantigens
- Induction of T_h2 responses to soluble proteins is independent of B cell tolerance status
- FcR cross-linking on monocytes results in impaired T cell stimulatory capacity
- A model for antigen-induced T cell unresponsiveness based on autophosphorylative protein tyrosine kinase activity