Complement peptide C3a inhibits IgE-mediated triggering of rat mucosal mast cells
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概要
著者
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PECHT Israel
Department of immunology, The Weizmann Institute of Science
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Pecht Israel
Department Of Chemical Immunology The Weizmann Institute Of Ccience
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ERDEI Anna
Department of Immunology, Eotvos Lorand University
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Erdei Anna
Department Of Immunology Eotvos Lorand University
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ANDREEV Sergey
Reseach Institute of highly Pure Biochemicals,Pudozhskaya 7
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Andreev Sergey
Reseach Institute Of Highly Pure Biochemicals Pudozhskaya 7
関連論文
- Binding of copolymer 1 and myelin basic protein leads to clustering of class II MHC molecules on antigen-presenting cells
- Bone marrow-derived mast cell differentiation is strongly reduced in histidine decarboxylase knockout, histamine-free mice
- C3a-derived peptide binds to the type I FcεR and inhibits proximal-coupling signal processes and cytokine secretion by mast cells
- Complement peptide C3a inhibits IgE-mediated triggering of rat mucosal mast cells
- A further link between innate and adaptive immunity: C3 depositon on antigen-presenting cells enhances the proliferation of antigen-specific T cells
- Modeling the presentation of C3d-coated antigen by B lymphocytes : enhancement by CR1/2-BCR co-ligation is selective for the co-ligating antigen
- Multiple synergizing factors contribute to the strength of the CD8^+ T cell response against listeriolysin O