Are CD4^+ T_h1 cells pro-inflammatory of anti-inflammatory? The ratio of IL-10 o IFN-γ or IL-2 determines thiir function
スポンサーリンク
概要
著者
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Katsikis Peter
Department Of Genetics Beckman Center B007 Stanford University School Of Medicine
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Katsikis Peter
Department Of Genetics Stanford University
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FELDMANN Marc
Kennedy Institute of Rheumatology Division, Imperial College School of Medicine
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Feldmann Marc
Kennedy Institute Of Rheumatology Sunley Division
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CHOEN Shara
Kennedy Institute of Rheumatology, Sunley Division
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LONDEI Marco
Kennedy Institute of Rheumatology, Sunley Division
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Choen Shara
Kennedy Institute Of Rheumatology Sunley Division
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Londei Marco
Kennedy Institute Of Rheumatology Sunley Division
関連論文
- HIV type 1 Tat protein enhances activation-but not Fas (CD95)-induced peripheral blood T cell apoptosis in healthyindividuals
- IL-15 enhances the function and inhibits CD95/Fas-induced apoptosis of human CD4^+ and CD8^+ effector-memory T cells
- Activation-induced peripheral blood T cell apoptosis is Fas independent in HIV-infected individuals
- Effective antigen presentation by dendritic cells is NF-κB dependent : coordinate regulation of MHC, co-stimulatory molecules and cytokines
- Are CD4^+ T_h1 cells pro-inflammatory of anti-inflammatory? The ratio of IL-10 o IFN-γ or IL-2 determines thiir function
- Comparison of peptide and suqerantigen-induced anergy in a peptide-specific polyclonal human T cell line