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First Department of Internal Medicine, Sapporo Medical University | 論文
- Upregulation of BNIP3 by 5-aza-2'-deoxycytidine sensitizes pancreatic cancer cells to hypoxia-mediated cell death
- DNA methylation changes in gastrointestinal disease
- Thymidine phosphorylase expression in human colorectal mucosa, adenoma and carcinoma : Role of p53 expression
- Acinar Cell Carcinoma of the Pancreas with Intraductal Growth : Report of a Case
- Expression of an Immunotherapeutic Target Antigen 17-1A for Colorectal Cancer in Multiple Myeloma
- MUC1 AS A TARGET ANTIGEN
- Enhancement of reactivity of anti-MUC1 core protein antibody and killing activity of anti-MUC1 cytotoxic T cell by deglycosylation of target tissues or cells
- Overexpression of cyclin D1 in chronic lymphoproliferation with t(11;14)(q13;32)
- Idiopathic CD4^+ T-Lymphocytopenia with Bowen's Disease
- Cytotoxic T lymphocytes derived from bone marrow mononuclear cells of multiple myeloma patients recognize an underglycosylated form of MUC1 mucin
- Isolation and characterization of a human cDNA encoding a protein homologous to the 7.2-kDa protein (subunit X) of bovine ubiquinol-cytochrome C reductase
- An Acromegalic Patient with Pulsatile Secretion of Growth Hormone (GH) Coincident with the Slow-Wave Sleep
- A new conceptualization for Mikulicz's disease as an IgG4-related plasmacytic disease
- INSULIN-LIKE GROWTH FACTOR-I RECEPTOR AS A CANDIDATE FOR A NOVEL MOLECULAR TARGET IN GASTROINTESTINAL CANCERS
- STUDY OF THE TUMOR VESSELS IN DEPRESSED-TYPE EARLY GASTRIC CANCERS USING NARROW BAND IMAGING MAGNIFYING ENDOSCOPY AND cDNA ARRAY ANALYSIS
- The expression of matrix metalloproteinase matrilysin indicates the degree of inflammation in ulcerative colitis
- Increased cyclooxygenase-2 expression in large flat colorectal tumors (laterally spreading tumors)
- Analysis of gene expression in human colorectal cancer tissues by cDNA array
- Matrix Metalloproteinase Matrilysin in Cancers of the Digestive Organs (Review)
- Review: The Discovery of the Widespread Microsatellite Instability Phenotype and Distinction of the Mutator and Suppressor Pathways for Gastrointestinal Cancer