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Division of Protein Biosynthesis, Institute for Protein Research, Osaka University | 論文
- Regulation of Reactive Oxygen Species by Nerve Growth Factor but not Bcl-2 as a Novel Mechanism of Protection of PC12 Cells from Superoxide Anion-Induced Death
- Free Radical-Independent Protection by Nerve Growth Factor and Bcl-2 of PC12 Cells from Hydrogen Peroxide-Triggered Apoptosis
- Repair of Chromosomal Damage : Transcription-Coupled DNA Repair and Its Disorders(Symposium : Manipulating Chromosomes)(Abstracts of the 52nd Annual Meeting of the Society of Chromosome Research)
- Analysis of Tyrosine Phosphorylation-Dependent Protein-Protein Interactions in TrkB-Mediated Intracellular Signaling Using Modified Yeast Two-Hybrid System^1
- Comparison of Survival-Promoting Effects of Brain-Derived Neurotrophic Factor and Neurotrophin-3 on PC12h Cells Stably Expressing TrkB Receptor^1
- MS-430, a Synthetic Pyrimidine Derivative, Influences the Intracellular Signal transduction Pathway Leading to Neuronal Differentiation of PC12h Cells
- Apoptosis-Signal Regulating Kinase-1 Is Involved in the Low Potassium-Induced Activation of p38 Mitogen-Activated Protein Kinase and c-Jun in Cultured Cerebellar Granule Neurons
- Brain-derived neurotrophic factor prevents superoxide anion-induced death of PC12h cells stably expressing TrkB receptor via modulation of reactive oxygen species
- Interaction of Ganglioside with Specific Peptide Sequences as a Mechanism for the Modulation of Calmodulin-Dependent Enzymes
- Role of Neurotrophin in Low K^+-Mediated Apoptosis of Cultured Cerebellar Granule Neurons
- WA8242A_1, A_1 and B, Novel Secretory Phospholipase A_1 Inhibitors Produced by Streptomyces violaceusniger I. Taxonomy, Production and Purification
- Generation of Free Radicals during the Death of Saccharomyces cerevisiae Caused by Lipid Hydroperoxide
- Survival of and 1-methyl-4-phenylpyridinium (MPP^+) neurotoxicity against dopaminergic neurons in coculture of rat mesencephalon with their target of non-target regions
- Regulation of Reactive Oxygen Species by Nerve Growth Factor but not Bcl-2 as a Novel Mechanism of Protection of PC12 Cells from Superoxide Anion-Induced Death