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Departments Of Allergy And Rheumatology University Of Tokyo Graduate School Of Medicine | 論文
- PJ-626 The anti-60kDa Heat-Shock Protein IgM Antibody as a Pathophygiological Candidate after the Repetitive Coxsackievirus Infection.(Cardiomyopathy, Basic and Clinical 2 (M) : PJ105)(Poster Session (Japanese))
- Severe airflow limitation in two patients with systemic lupus erythematosus : effect of inhalation of anticholinergics
- Wegener's Granulomatosis with Eosinophilia
- Effects of In Vivo Gene Transfer of Fibroblast Growth Factor-2 on Cardiac Function and Collateral Vessel Formation in the Microembolized Rabbit Heart
- Guillain-Barre syndrome following herpes zoster in a patient with systemic sclerosis
- Association of a Cellular 21-kDa Transmembrane Protein (VAP21) with Enveloped Viruses
- Accumulation of Identical T Cell Clones in the Right and Left Lobes of the Thyroid Gland in Patients with Graves' Disease : Analysis of T Cell Clonotype in vivo
- CrossLinking of the CD69 Molecule Enhances S100A9 Production in Activated Neutrophils
- Proteomic Surveillance of Autoantigens in Relapsing Polychondritis
- Identification of Autoantibodies With the Corresponding Antigen for Repetitive Coxsackievirus Infection-Induced Cardiomyopathy
- IL-33-induced activation of human basophils and eosinophils via ST2
- Human Basophils and Cytokines/Chemokines
- Three Cases of Ortho-phthalaldehyde-induced Anaphylaxis after Laryngoscopy : Detection of Specific IgE in Serum
- Trans-basement membrane migration of human basophils : role of matrix metalloproteinase-9
- IgE- and FcεRI-mediated migration of human basophils
- Vasculo-Behcet's disease with non-traumatic subcapsular hematoma of the kidney and aneurysmal dilatations of the celiac and superior mesenteric arteries
- An efficient method to prepare T cell receptor gene-transduced cytotoxic T lymphocytes type 1 applicable to tumor gene cell-therapy
- T_h -derived cytokine IFN-γ is a potent inhibitor of eotaxin synthesis in vitro
- Antigen speciflc T cells transduced with interleukin-10 ameliorate experimentally induced arthritis without impairing systemic immune response to the antigen
- Autoreactive T cells as a therapeutic vehicle