スポンサーリンク
Department of Dermatology, Keio University School of Medicine | 論文
- Two Cases of Atypical Bullous Disease Showing Linear IgG and IgA Deposition in the Basement Membrane Zone
- He II Thermal Counterflow near Superfluid Turbulent Transition : I. QUANTUM LIQUIDS AND SOLIDS : Superfluid Flow
- Solubility of ^4He in Liquid ^3He at Very Low Temperatures
- Solubility of ^4He in Liquid ^3He at Very Low Temperatures : I. QUANTUM LIQUIDS AND SOLIDS : Liquid ^4He and ^3He-^4He
- Immune dysregulation of pemphigus in humans and mice
- Melanogenesis, biosynthetic phenotype of fibronectin and collagen, and migrating activity in cloned B16 mouse melanoma cells
- Predominant IgG4 subclass in autoantibodies of pemphigus vulgaris and foliaceus
- Trichophyton Tonsurans Infection Manifesting as Multiple Concentric Annular Erythemas
- Thymoma with Pemphigus Foliaceus
- Experimental evaluation of ebastine, a second-generation antihistamine, as a supportive medication for alopecia areata
- Tranilast inhibits collagen synthesis in normal, scleroderma and keloid fibroblasts at a late passage culture but not at an early passage culture
- Photoluminescence Enhancement of ZnS:Mn^ Nanocrystal Phosphors : Comparison of Organic and Inorganic Surface Modifications
- No activation of urokinase plasminogen activator by anti-desmoglein 3 monoclonal IgG antibodies in cultured human keratinocytes
- Genotype-phenotype correlations in six Japanese patients with recessive dystrophic epidermolysis bullosa with the recurrent p.Glu2857X mutation
- Autoimmune and infectious skin diseases that target desmogleins
- Staphylococcal exfoliative toxins : "Molecular scissors" of bacteria that attack the cutaneous defense barrier in mammals
- Plasma Polymerization for Protein Patterning : Reversible Formation with Fullerence Modification
- Pemphigus as a paradigm of autoimmunity and cell adhesion
- Removal of amino-terminal extracellular domains of desmoglein 1 by staphylococcal exfoliative toxin is sufficient to initiate epidermal blister formation
- Subcellular localization of desmosomal components is different between desmoglein3 knockout mice and pemphigus vulgaris model mice