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Department of Cardiology, Iizuka hospital | 論文
- Clinical characteristics and outcomes of heart failure with preserved ejection fraction : Lessons from epidemiological studies
- Colestimide, an anion exchange resin agent, can decrease the number of LDL particles without affecting their size in patients with hyperlipidemia
- Direct Inhibition of N-Methyl-D-Aspartate (NMDA)-Receptor Function by Antiglaucomatous β-Antagonists
- PE-433 Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthase Isoforms(Diabetes/Obesity/Metabolic syndrome-4 (H) PE73,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- OE-199 Genetic Disruption of All Nitric Oxide Synthase Isoforms Markedly Reduces Survival in Mice(Endothelium-1 (H) OE34,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- OE-133 Vasculoprotective Role of the Nitric Oxide Synthases System against Vascular Lesion Formation in Mice in Vivo(Peripheral circulation-1 (H) OE23,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation S
- OE-063 Spontaneous Development of Systemic Arteriosclerosis in Mice Lacking All Nitric Oxide Synthase Isoforms(Atherosclerosis, basic-1 (H) OE11,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society
- FRS-084 Spontaneous Myocardial Infarction in Mice Lacking All Nitric Oxide Synthase Isoforms(Molecular Biology in Cardiovascular Diseases (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- FRS-021 Long-Term Treatment with an Angiotensin II Type 1 Receptor Blocker Ameliorates Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthases(Diabetes/Obesity (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- Development of Genetically Engineered Mice Lacking All Three Nitric Oxide Synthases
- Granulocyte Activation in Restenosis After Percutaneous Transluminal Coronary Angioplasty
- OE-221 Statins Reduce Interleukin (IL)-1 β and IL-6-Induced Plasminogen Activator Inhibitor-1 in Hepatocytes Through C/EBPδ(Thromboembolism/Antithrombotic therapy/Thrombolysis-1 (H) OE37,Oral Presentation (English),The 70th Anniversary Annual Scientific M
- Atorvastatin Upregulates Vascular Neuronal Nitric Oxide Synthase Expression Through the Akt/NF-κB Pathway(Endothelium/NO 4 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Vascular Neuronal Nitric Oxide Synthase Expression is Upregulated in Response to Platelet-Derived Growth Factor Through the MEK/ERK Pathway(Endothelium/NO 4 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Neuronal Nitric Oxide Synthase Is Subject to Expressional Regulation by Inflammatory Stimuli in Vascular Smooth Muscle Cells in Rats
- Blockade of Matrix Metalloproteinase (MMP)-2 Exacerbates Myocardial Inflammation and Dysfunction in Tumor Necrosis Factor (TNF)-α-Induced Cardiomyopathy(Heart Failure, Basic 1 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Overexpression of Mitochondrial Transcription Factor A Prevents Mitochondrial Deficiencies and Cardiac Failure after Myocardial Infarction(Clinical and Basic Research in Heart Failure (M), The 69th Annual Scientific Meeting of the Japanese Circulation Soc
- OE-112 Inhibition of Transforming Growth Factor (TGF)-β Signaling Prevents Left Ventricular Remodeling and Failure after Myocardial Infarction(Heart Failure, Basic 1 (M) : OE13)(Oral Presentation (English))
- Inhibition of Transforming Growth Factor (TGF)-β, Signaling Exacerbates Early Left Ventricular Failure after Acute Myocardial Infarction
- OJ-163 Genetic Disruption of All Nitric Oxide Synthase Isoforms Abolishes EDHF-Mediated Responses in Mice(Endothelium-2 (H) OJ28,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
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