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Department Of Pharmacology Setsunan University | 論文
- Endogenous and Exogenous Glucocorticoids Prevent Trimethyltin From Causing Neuronal Degeneration of the Mouse Brain In Vivo : Involvement of Oxidative Stress Pathways
- Activation of c-Jun N-Terminal Kinase Cascades Is Involved in Part of the Neuronal Degeneration Induced by Trimethyltin in Cortical Neurons of Mice
- Transcriptional Factors in the Cochlea Within the Inner Ear
- CHARACTERIZATION OF Na^+-DEPENDENT BINDING OF [^3H] GLUTAMATE IN CEREBRAL SYNAPTIC MEMBRANES
- Gene Transcription Through Myc Family Members in Eukaryotic Cells
- Possible Involvement of Membrane Phospholipids in Inhibition by Ferrous Ions of [^3H]MK-801 Binding to the Native N-Methyl-D-Aspartate Channel in Rat Brain
- Immunochemical Studies of Exocellular Polysaccharides of Tremella and Cryptococcus(Biological Chemistry)
- Adult Neurogenesis Is Regulated by Endogenous Factors Produced During Neurodegeneration
- Endogenous Nitric Oxide Generation Linked to Ryanodine Receptors Activates Cyclic GMP/Protein Kinase G Pathway for Cell Proliferation of Neural Stem/Progenitor Cells Derived From Embryonic Hippocampus
- Indomethacin Ameliorates Trimethyltin-Induced Neuronal Damage in Vivo by Attenuating Oxidative Stress in the Dentate Gyrus of Mice