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Department Of Pharmacology Graduate School Of Pharmaceutical Sciences Hokkaido University | 論文
- 2-(3-Pyridyl)thiazolidine-4-carboxamide Derivatives. II. Structure-Activity Relationships and Active Configuration of 2-(3-Pyridyl)thiazolidine-4-carboxamides as Platelet-Activating Factor Receptor Antagonists
- 2-(3-Pyridyl)thiazolidine-4-carboxamides. 1. Novel Orally Active Antagonists of Platelet-Activating Factor (PAF)
- Age-Related Alterations in the Expression of Glial Cell Line-Derived Neurotrophic Factor in the Senescence-Accelerated Mouse Brain
- Inhibition of glutamatergic transmission by morphine in the basolateral amygdaloid nucleus reduces pain-induced aversion
- Antibacterial activity of traditional medicines and an active constituent lupulone from Humulus lupulus against Helicobacter pylori
- TEK/Tie2 Is a Novel Gene Involved in Endoplasmic Reticulum Stress
- PI3K-Akt inactivation induced CHOP expression in endoplasmic reticulum-stressed cells
- Alteration of Leptin-Induced STAT3 Activation in the Brain of Senescence-Accelerated Mouse (SAM) P8(Pharmacology)
- Aspirin and Sodium Salicylate Protect Brain Cell Death Against Endoplasmic Reticulum Stress
- Possible Role of Interleukin-6 in PC12 Cell Death Induced by MPP^+ and Tetrahydroisoquinoline
- Cholecystokinin Inhibits Food Intake Independent of Interleukin-1β Expression in the Brain(Pharmacology)
- Age-Related Alterations in the Expression of Glial Cell Line-Derived Neurotrophic Factor in the Senescence-Accelerated Mouse Brain
- The Age-Related Degeneration of Oligodendrocytes in the Hippocampus of the Senescence-Accelerated Mouse (SAM) P8 : A Quantitative Immunohistochemical Study(Pharmacology)
- Manganese Superoxide Dismutase Is Induced by Endoplasmic Reticulum Stress through IRE1-Mediated Nuclear Factor (NF)-κB and AP-1 Activation(Biochemistry/Molecular Biology)
- Effects of S-Nitroso-Cysteine on Proteins That Regulate Exocytosis in PC12 Cells: Inhibitory Effects on Translocation of Synaptophysin and ADP-Ribosylation of GTP-Binding Proteins
- Stimulation of Noradrenaline Release by T-588, a Cognitive Enhancer, in PC12 Cells
- The Effects of T-588, a Novel Cognitive Enhancer, on Noradrenaline Uptake and Release in Rat Cerebral Cortical Slices
- ATP Receptor-Mediated Increase of Ca Ionophore-Stimulated Arachidonic Acid Release from PC12 Pheochromocytoma Cells
- Effects of Protein Kinase C and A Activation on ATP-Stimulated Release of [^3H] Noradrenaline from PC12 Cells
- Brain Cytokines and Chemokines : Roles in Ischemic Injury and Pain