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Department Of Medicine Ii Kansai Medical University | 論文
- PE-221 Can We Predict The Presence of Significant Coronary Stenosis by Distribution of Vascular or Valvular Calcification With MDCT?(CT/DSA-05, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- PE-341 The Longitudinal Calcification Length in Multi-slice Computed Tomography May be Available to Detect the Coronary Artery Stenosis(CT/DSA-5 (I) PE57,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation S
- PE-317 Angiotensin Receptor 1 Blocker Inhibits Atherosclerotic Changes of Aortic Valve by Preserving Endothelial Cells and Inhibiting Transformation into Osteoblast Phenotype(Vascular heart disease/Pericarditis/Cardiac tumor-2 (M) PE53,Poster Session (Eng
- PE-047 Angiotensin II Type1 Receptor Blocker, Olmesartan Inceases Plaque Stability with Altering Macrophage Activation in Rabbit Plaque Model(Atherosclerosis, basic-3 (H) PE8,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the J
- PJ-131 Bone Marrow AT1 and AT2 Modulate Vascular Repair by Regulating Mobilization and Differentiation of Vascular Progenitor Cells and Macrophage Activation(Restenosis basic/clinical-3, The 71st Annual Scientific Meeting of the Japanese Circulation Socie
- PJ-072 Angiotensin II Receptor Blocker Stabilizes Vulnerable Plaque and Reduces Susceptibility of Plaque Rupture in Apolipoprotein E Deficient Mice(Atherosclerosis, basic-8, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-068 A Wire-Induced Arterial Injury Accelerated the Destabilization of Remote Established Atherosclerotic Plaques in ApoE-deficient Mice(Atherosclerosis, basic-8, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- PE-034 Bone Marrow AT1 Accelerated the Development of Atherosclerosis by Regulating Differentiation of Macrophage Progenitors and Macrophage Functions(Atherosclerosis, basic-4, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- OJ-026 Angiotensin II Type2 Receptor in Bone Marrow-Derived Cells Inhibits Neointimal Hyperplasia by Counteracting Type1 Receptor Function in Mouse Wire-Injured Artery(Restenosis, basic/clinical-1 (IHD) OJ5,Oral Presentation (Japanese),The 70th Anniversar
- OE-201 Erythropoietin-Mediated Mobilization of Endothelial Progenitors Enhances Reendothelialization via Akt-eNOS activation and Prevents Neointimal Hyperplasia of Injured Arteries(Endothelium-1 (H) OE34,Oral Presentation (English),The 70th Anniversary An
- FRS-002 Angiotensin II Type 1 Receptor in Bone Marrow-Derived Stem Cells Plays a Crucial Role in Atherosclerosis Development(Frontier of Atherosclerosis Research (H) FRS1,Featured Research Session,The 70th Anniversary Annual Scientific Meeting of the Japa
- Erythropoietin Accelerates Regeneration of Endothelium and Prevents Neointimal Hyperplasia after Vascular Injury by Mobilizing Endothelial Progenitor Cells(Endothelium/NO 4 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- IS070 Angiotensin Type 2 Receptors Are Re-expressed by Cardiac Fibroblasts from Failing Myopathic Hamster Hearts and Inhibit Cell Growth and Fibrillar Collagen Metabolism
- IS050 Cardiac-Specific Overexpression of Angiotensin AT2 receptor Causes Attenuated Response to AT1 receptor - Mediated Chronotropic Action
- Down-Regulation by cAMP of Angiotensin II Type 2 Receptor Gene Expression in PC12 Cells
- PE-402 Obesity: an Independent Risk for the Chronic Kidney Disease(PE068,Obesity/SAS 2 (H),Poster Session (English),The 73rd Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-639 Pitavastatin Increases Circulating Adiponectin in Hyperlipemic Patients(Atherosclerosis, clinical-13 (H) PJ107,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- Relation Selecins and Chemokines in Patients after Coronary Angioplasty
- The effect of loading condition and heart rate on electrophysiological status in situ canine hearts
- Enhancement of Ischemia-Induced Angiogenesis by eNOS Overexpression
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