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Department Of Cardiovascular Medicine The University Of Tokyo:presto Science And Technology Agency | 論文
- Histopathological Reaction of a Calcium Phosphate Cement Root Canal Filler
- Adipose Tissue Remodeling and Malfunctioning in Obesity Revealed by in vivo Molecular Imaging
- FRS-060 SUMOylation of KLF5 is a key molecular switch of transcriptional regulatory programs of lipid metabolism(New Insights into Metabolic Syndrome(02)(H),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- OE-174 Molecular Mechanism of Soluble Form of VEGFR-1 (sVEGFR-1)Expression in Vascular Endothelial Cell(Molecular biology, vascular(01)(IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- OE-150 Complex structure of ventricular wall modulates the globally applied stretch to create heterogeneous strain distribution and trigger the fatal arrhythmia(Arrhythmia, basic(01)(A),Oral Presentation(English),The 72nd Annual Scientific Meeting of the
- Obese adipose tissue remodeling, malfunctioning, and chronic inflammation visualized by in vivo molecular imaging
- PJ-178 Free Fatty Acid, Palmitate, Induces Metabolic and Vascular Lipotoxicity via the IRE1 Arm of ER Stress Response Pathways(PJ030,Metabolic Syndrome 1 (H),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
- 4 Bone marrow-derived cells contribute to vascular wall inflammation without differentiating into smooth muscle cells(Young Investigator's Award Finalists Lectures (YIA),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
- Acute onset of nephrotic syndrome during interferon-α retreatment for chronic active hepatitis C
- Drug-induced MPO-ANCA-positive necrotizing crescentic glomerulonephritis preceded by granulomatous hepatitis
- DPE-071 Retinoic Acid Receptor Antagonist Ameliorates Pressure Overload-Induced Cardiac Hypertrophy and Remodeling(DPE12,Cardiomyopathy/Hypertrophy (Basic) (M),Digital Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation
- OE-057 Saturated Fatty Acid, Palmitate, Promotes Smooth Muscle Phenotypic Modulation and Exacerbates Neointima Formation(OE10,Atherosclerosis, Basic 1 (IHD),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society
- FRS-104 KLF5 Controls Tissue Remodeling in Cardiovascular and Kidney Diseases via S100 Proteins(FRS21,New Insights into Chronic Kidney Disease and Renal Circulation (H),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japanese
- FRS-093 Significance of the Transcription Factor KLF5 in Myocardial Hypertrophy in Response to Pressure Overload(FRS19,Genetic Abnormalities in Cardiac Hypertrophy and Cardiomyopathy (M),Featured Research Session (English),The 73rd Annual Scientific Meeti
- PE-183 KLF5 Plays an Important Role in Endoplasmic Reticulum Stress Response in both Cardiovascular and Metabolic Systems(Diabetes/Obesity/Metabolic syndrome(05)(H),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Soc
- PE-030 Endoplasmic Reticulum Stress Signaling Modulates Smooth Muscle Phenotypes(Atherosclerosis, basic(03)(IHD),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- OE-256 Significance of the Transcription Factor KLF5 in Myocardial Hypertrophy in Response to Pressure Overload(Cardiac hypertrophy, basic/clinical(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- OE-235 Identification of Metabolic Syndrome Related-unknown Key Molecule With Using siRNA Library(Metabolism/Biochemistry/Energetics(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- FRS-093 KLF5 controls fibrosis in cardiovascular and kidney diseases via S100 protein expression(Renal Dysfunction and Cardiovascular Disease(H),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- PE-539 S100A4 is induced by Angiotensin II in Fibrosis and the Target of Beraprost(Molecular biology, vascular-3, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
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