Kodaka Tetsuro | 理化学研究所横浜研究所免疫アレルギー科学総合研究センター 自己免疫制御研究グループ
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概要
関連著者
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Kodaka Tetsuro
理化学研究所横浜研究所免疫アレルギー科学総合研究センター 自己免疫制御研究グループ
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Kodaka Tetsuro
Laboratory For Autoimmune Regulation Riken Center For Allergy And Immunology Riken Yokohama Institut
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KATO Takako
Laboratory of Molecular Biology and Gene Regulation, Department of Life Science, School of Agricultu
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MORI Yoshiko
Laboratory for Autoimmune Regulation, RIKEN Center for Allergy and Immunology, RIKEN Yokohama Instit
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KANAGAWA Edith
Laboratory for Autoimmune Regulation, RIKEN Center for Allergy and Immunology, RIKEN Yokohama Instit
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KANAGAWA Osami
Laboratory for Autoimmune Regulation, RIKEN Center for Allergy and Immunology, RIKEN Yokohama Instit
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Mori Yoshiko
Laboratory For Autoimmune Regulation Riken Center For Allergy And Immunology Riken Yokohama Institut
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Kanagawa Edith
Laboratory For Autoimmune Regulation Riken Center For Allergy And Immunology Riken Yokohama Institut
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Kanagawa Osami
Laboratory For Autoimmune Regulation Riken Center For Allergy And Immunology Riken Yokohama Institut
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Kanagawa Edith
Laboratory For Autoimmune Regulation Research Center For Allergy And Immunology Riken Yokohama Insti
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Kato Takako
Laboratory For Autoimmune Regulation Research Center For Allergy And Immunology Riken Yokohama Insti
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Kodaka Tetsuro
Laboratory For Autoimmune Regulation Research Center For Allergy And Immunology Riken Yokohama Insti
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Mori Yoshiko
Laboratory For Autoimmune Regulation Research Center For Allergy And Immunology Riken Yokohama Insti
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Kanagawa Osami
Laboratory For Autoimmune Regulation Research Center For Allergy And Immunology Riken Yokohama Insti
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HORI Shohei
Research Unit for Immune Homeostasis, RIKEN Center for Allergy and Immunology, RIKEN Yokohama Instit
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Hori Shohei
Research Unit For Immune Homeostasis Riken Center For Allergy And Immunology Riken Yokohama Institut
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Hori Shohei
Research Unit For Immune Homeostasis Research Center For Allergy And Immunology
著作論文
- Protection of IFN-γ signaling-deficient NOD mice from diabetes by cyclophosphamide
- Critical role of IFN-γ in CFA-mediated protection of NOD mice from diabetes development