Yorifuji T | Department of Pediatrics, Kyoto University School of Medicine
スポンサーリンク
概要
関連著者
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Kawai M
Department Of Applied Chemistry Nagoya Institute Of Technology
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Yorifuji T
Department Of Baioscience And Biotechnology Shinshu University
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Yorifuji T
Department Of Pediatrics Kyoto University School Of Medicine
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Momoi T
First Department of Pediatrics, Japanese Red Cross Society Wakayama Medical Center
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Kawai M
Department Of Pediatrics Kyoto University School Of Medicine
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Yorifuji T
Department of Pediatrics, Kyoto University School of Medicine
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Nakahata T
Department Of Pediatrics Hirosaki University School Of Medicine
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Kurokawa K
Department Of Anatomy Shiga University Of Medical Science
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Mamada M
Department of Pediatrics, Kyoto University Hospital
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Momoi T
Department of Pediatrics. Japanese Red Cross Society Wakayama Medical Center
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Nakahata T
Department Of Pediatrics Kyoto University Hospital
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Kurokawa K
Department Of Pediatrics Kyoto University Hospital
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Mamada M
Department Of Pediatrics Kyoto University Hospital
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Katsumata N
Department Of Endocrinology And Metabolism National Research Institute For Child Health And Developm
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Ishikawa H
Department Of Anatomy Gunma University School Of Medicine
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Tanaka R
First Department of Pediatrics, Japanese Red Cross Society Wakayama Medical Center
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Muroi J
Department of Pediatrics, Kyoto University Hospital
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Momoi T
First Department Of Pediatrics Japanese Red Cross Society Wakayama Medical Center
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Katsumata N
Department Of Endocrinology And Metabolism National Research Institute For Child Health And Developm
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Muroi J
Department Of Pediatrics Kyoto University Hospital
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Katsumata N
Department Of Endocrinology And Metabolism National Research Institte For Child Health And Developme
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Kinoshita E
Department Of Pediatrics Nagasaki University School Of Medicine
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Kinoshita E
Department Of Pediatrics Nagasaki University Graduate School Of Biomedical Sciences
著作論文
- 23 A CASE OF FOCAL NESIDIOBLASTOSIS TREATED WITH PARTIAL PANCREATECTOMY
- 74 AN ASSOCIATION OF THE FBN1 GENE POLYMORPHISM WITH TALL STATURE
- 72 A NONSENSE MUTATION IN EXON 3 FO THE GH1 GENE CAUSE FAMIKIAK ISOLATED GROWTH HORMONE DEFICIENCY TYPE II
- 71 A NONSENSE MUTATION IN EXON 3 OF THE GH1 GENE CAUSES FAMILIAL ISOLATED GROWTH HORMONE DEFICIENCY TYPE II
- 114 A CASE OF DOWN SYNDROME WITH IDIOPATHIC HYPERCALCEMIA
- 168 A frameshift mutation and a familial deletion mutation of the DAX1 gene in patients with X-linked adrenal hypoplasia congenita