Kaneda R. | Institute of Immunological Science, Hokkaido University

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概要

• Kaneda R.の詳細を見る
• 同名の論文著者
• Institute of Immunological Science, Hokkaido Universityの論文著者

関連著者

• Onoe K.

  Institute Of Immunological Science Hokkaido University

• Kaneda R.

  Institute of Immunological Science, Hokkaido University

• Kaneda R.

  Institute Of Immunological Science Hokkaido University

• Iwabuchi K.

  Institute of Immunological Science, Hokkaido University

• Iwabuchi K.

  Division Of Immunobiology Institute For Genetic′ Medicine Hokkaido University

• Kajiwara M.

  Institute Of Immunological Science Hokkaido University

• Kasai M.

  National Institute Of Health

• Iwabuchi K.

  Institute Of Immunological Science Hokkaido University

• IWABUCHI K.

  Institute for Environmental and Gender-specific Medicine, Juntendo University Graduate School of Medicine

• Onoe K.

  ′ Section Of Pathology Institute Of Immunological Science ′ Hokkaido University′ Hokkaido University

• Iwabuchi K.

  Section Of Pathology Institute Of′ Immunological Science Hokkaido University

• Itoh Y.

  Institute of Immunological Science, Hokkaido University

• Onoe K.

  ′ Section Of Pathology Institute Of Immunological Science ′ Hokkaido University′ Hokkaido University

• Murakami M.

  Institute of Laser Engineering, Osaka University

• Iwabuchi K

  Institute of Immunological Science, Hokkaido University

• Katoh M.

  Department Of′ Bacterial And Blood Products National Institute Of Infectious′ Diseases

• Uede T.

  Institute of Immunological Science, Hokkaido University

• Iwabuchi K

  Institute Of Immunological Science Hokkaido University

• Uede T.

  Institute Of Immunological Science Hokkaido University

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著作論文

• Dissociation of Fas-mediated cytotoxicity and Fas L expression in a cytotoxic CD4+ T cell clone - Comparative analysis of Fas-mediated cytotoxicity between a T hybridoma and a T cell clone-.
• Detachment activity but not cytotoxicity induced in a T cell clone following antigen presentation in the presence of thymic epithelial cells.
• Selective antigen presenting activity of cortical thymic epithelial cells against CD4+ T cells associated with both lack of costimulatory molecules and inefficient presentation of MHC-peptide ligands.

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