Ohigashi Makoto | Department Of Clinical Pharmacology Division Of Pathological Science Kyoto Pharmaceutical University
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概要
- Ohigashi Makotoの詳細を見る
- 同名の論文著者
- Department Of Clinical Pharmacology Division Of Pathological Science Kyoto Pharmaceutical Universityの論文著者
関連著者
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Katamura Maki
AMI-Kyoto Multi-Center Risk Study Group
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Nakamura T
Second Department Of Internal Medicine Kyoto Prefectural University Of Medicine
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Kobara Miyuki
Department of Cardiology, Kyoto Prefectural University School of Medicine
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Katamura Maki
Department Of Cardiology Kyoto Second Red Cross Hospital
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Nakata Tetsuo
Department Of Cardiovascular Medicine Nantan General Hospital
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TOBA Hiroe
Department of Clinical Pharmacology, Kyoto Pharmaceutical University
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Toba Hiroe
Department Of Clinical Pharmacology Division Of Pathological Science Kyoto Pharmaceutical University
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Tanaka Kenta
Department Of Clinical Pharmacology Division Of Pathological Science Kyoto Pharmaceutical University
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Kobara Miyuki
Departments Of Cardiovascular Medicine Kyoto Prefectural University School Of Medicine
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Kobara Miyuki
Department Of Clinical Pharmacology Kyoto Pharmaceutical University
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Kobara Miyuki
Department Of Cardiovascular Medicine Kyoto Prefectural University School Of Medicine
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Ohigashi Makoto
Department of Clinical Pharmacology, Division of Pathological science, Kyoto Pharmaceutical Universi
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Uchida Yuugo
Department of Clinical Pharmacology, Division of Pathological science, Kyoto Pharmaceutical Universi
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Uchida Yuugo
Department Of Clinical Pharmacology Division Of Pathological Science Kyoto Pharmaceutical University
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Nomura Tetsuya
Department Of Cardiovascular Medicine Kyoto Prefectural University School Of Medicine
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Tanaka Kenta
Department Of Applied Chemistry School Of Engineering Osaka City University
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Kimata Masaki
Department Of Cardiology Kyoto First Red Cross Hospital
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Nakata Tetsuo
Department Of Cardiovascular Medicine Kyoto Prefectural University Of Medicine
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Kobara Miyuki
Department Of Cardiology Kyoto First Red Cross Hospital
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Nakata Tesuo
Department Of Clinical Pharmacology Kyoto Pharmaceutical University
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Nakata Tetsuo
Department Of Clinical Pharmacology Division Of Pathological Science Kyoto Pharmaceutical University
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Ohigashi Makoto
Department Of Clinical Pharmacology Division Of Pathological Science Kyoto Pharmaceutical University
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Nakamura Takeshi
Kyoto Prefectural University Of Medicine Department Of Cardiology
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Tanaka Kenta
Department of Applied and Bioapplied Chemistry, Graduate School of Engineering, Osaka City University
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Tanaka Nana
Department Of Clinical Pharmacology Kyoto Pharmaceutical University
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Matsubara Hiroaki
Department of Cardiovascular Medicine, Kyoto Prefectural University of Medicine
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Hayashi Hironori
Department Of Cardiovascular Medicine Kyoto Prefectural University Of Medicine
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Makino H
Department Of Medicine And Clinical Science Okayama University Graduate School Of Medicine And Denti
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Matsubara Hiroaki
Department Of Cardiology Kyoto Prefectural University Of Medicine
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ABE Masaki
Department of Clinical Pharmacology, Kyoto Pharmaceutical University
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Abe Masaki
Department Of Clinical Pharmacology Kyoto Pharmaceutical University
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Tsujimoto Shouta
Department of Clinical Pharmacology, Division of Pathological science, Kyoto Pharmaceutical Universi
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Matsuoka H
Kyoto Univ. Kyoto
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Abe Masaki
Department Of Agricultural Chemistry The University Of Tokyo
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Nakamura Takeshi
Department Of Cardiovascular Medicine Kyoto Prefectural University School Of Medicine
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Tsujimoto Shouta
Department Of Clinical Pharmacology Division Of Pathological Science Kyoto Pharmaceutical University
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Hayashi Hironori
Department Of Breast Oncology Aichi Cancer Center Hospital
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Matsubara Hiroaki
Department Of Bioscience National Cardiovascular Center Research Institute
著作論文
- FRS-072 Imatinib Induces Autophagy-Mediated Cell Death in Cardiac Myocytes(FRS15,Novel Molecular Mechanisms of Heart Failure 1 (M),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
- PJ-112 Aldosterone Activates Apoptosis Signal-Regulating Kinase 1 (ASK-1) through Nongenomical ROS production and Induces Myocyte Apoptosis.(Heart failure, basic(05)(M),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulatio