MURATA Yukie | Basic Research Laboratories, Ajinomoto Co. Inc.
スポンサーリンク
概要
関連著者
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Hamuro Junji
Basic Research Laboratories Ajinomoto Co. Inc.
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MURATA Yukie
Basic Research Laboratories, Ajinomoto Co. Inc.
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Murata Yukie
Basic Research Laboratories Ajinomoto Co. Inc.
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YAMASHITA Akira
Department of Biostatistics, Graduate School, Kansai Medical University
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SUGAMURA Kazuo
Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine
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Sugamura Kazuo
Department Of Immunology Tohoku University Graduate School Of Medicine
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Sugamura Kazuo
Department Of Immunology Tohoku University School Of Medicine
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SAITO Takashi
Division of Molecular Genetic, Center for Biomedical Science, School of Medicine, Chiba University
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Hamuro J
Ajinomoto Co. Inc. Kawasaki Jpn
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SHIMAMURA Toshiro
Basic Research Laboratories, Ajinomoto Co. Inc.
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SUZUKI Manabu
Basic Research Laboratories, Ajinomoto Co., Inc.
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TAKATSUKI Fumihiko
Basic Research Laboratories, Ajinomoto Co., Inc.
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SUGA Tetsuya
Basic Research Laboratories, Ajinomoto Co., Inc.
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Shimamura Toshiro
Basic Research Laboratories Ajinomoto Co. Inc.
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Takatsuki Fumihiko
Basic Research Laboratories Ajinomoto Co. Inc.
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Suga Tetsuya
Basic Research Laboratories Ajinomoto Co. Inc.
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Suzuki Manabu
Basic Research Laboratories Ajinomoto Co. Inc.
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Yamashita Akira
Department Of Anatomy Hamamatsu Medical University
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Saito Takashi
Division Of Cariology And Endodontology Department Of Oral Rehabilitation School Of Dentistry Health
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Saito T
Division Of Molecular Genetics Chiba University School Of Medicine
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Saito Takashi
Division for Behavior Analysis, Behavior Research Institute, Gunma University School of Medicine
著作論文
- The conversion of redox status of peritoneal macrophages during pathological progression of spontaneous inflammatory bowel disease in Janus family tyrosine kinase 3^ and IL-2 receptor γ^ mice
- The polarization of T_h1/T_h2 balance is dependent on the intracellular thiol redox status of macrophages due to the distinctive cytokine production
- THE TRIGGERING AND HEALING OF TUMOR STROMAL INFLAMMATORY REACTIONS REGULATED BY OXIDATIVE AND REDUCTIVE MACROPHAGES