KOLSCH Eckehart | Institut fur Immunologie,Domagkstrasse 3A
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概要
関連著者
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KOLSCH Eckehart
Institut fur Immunologie,Domagkstrasse 3A
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Kolsch Eckehart
Institut Fur Immunologie Domagkstrasse 3a
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Specht Christoph
Institut Fur Immunologie Universitat Munster
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Kolbe Ludger
Institut Fur Immunologie Domagkstrasse 3a
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Schmitt Edgar
Institut Fur Lmmunologie Obere Zahlbacher Strasse 67
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Rude Erwin
Institut Fur Lmmunologie Obere Zahlbacher Strasse 67
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Specht C
Institute For Immunology University Of Munster
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Gately Maurice
Department Of Inflammation/autoimmune Diseases Hoffmann-laroche Inc.
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Sudowe Stephan
Institut Fur Immunologie Universitat Munster
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GERMANN Tieno
Institut fur Immunologie,Obere Zahlbacher Strasse 67
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GUCKES Steffi
Institut fur Immunologie,Obere Zahlbacher Strasse 67
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BONGARTZ Martina
Institut fur Immunologie,Obere Zahlbacher Strasse 67
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DLUGONSKA Henryka
Institut fur Immunologie,Obere Zahlbacher Strasse 67
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KOLBE Ludger
Institut fur Immunologie,Domagkstrasse 3A
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PODLASKI Frank
Department of Inflammation/Autoimmune Diseases,Hoffmann-LaRoche Inc.
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CLEMENAS Andreas
Institute for Immunology, University of Munster
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RADEMAEKERS Andre
Institute for Immunology, University of Munster
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Germann Tieno
Institut Fur Lmmunologie Obere Zahlbacher Strasse 67
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Kolbe Ludger
Institut Fur Immunologie Universitat Munster
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Guckes Steffi
Institut Fur Immunologie Obere Zahlbacher Strasse 67
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Podlaski Frank
Department Of Inflammation/autoimmune Diseases Hoffmann-laroche Inc.
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Clemenas Andreas
Institute For Immunology University Of Munster
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Bongartz Martina
Institut Fur Immunologie Obere Zahlbacher Strasse 67
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Dlugonska Henryka
Institut Fur Immunologie Obere Zahlbacher Strasse 67
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Rademaekers Andre
Institute For Immunology University Of Munster
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Koelsch E
Institute For Immunology University Of Munster
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Kolsch Eckehart
Institut Fur Immunologie Universitat Munster
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Schmitt Edgar
Institut Fur Immunologie J. Gutenberg Universitat
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GERMANN Tieno
Institut Fur Immunologie, University of Mainz
著作論文
- Administration of IL-12 during ongoing immune responses fails to permanently suppress and can even enhance the synthesis of antigen-specific IgE
- The J558 V_H CDR3 region contributes little to antibody avidity; however, it is the recognition element for cognate T cell control of he α(1→3) dextran-specific antibody response
- IN situ dormancy of B lymphocytes programmed for an IgE antibody response and thier sudden release from unresponsiveness under in vitro conditions