Guo Feng | School of Pharmaceutical Science, China Medical University, China
スポンサーリンク
概要
関連著者
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WANG Wu-Yang
Department of Physiology, Graduate School of Medicine and Dental Sciences, Kagoshima University
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MINOBE Etsuko
Department of Physiology, Graduate School of Medicine and Dental Sciences, Kagoshima University
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XU Jian-Jun
Department of Physiology, Graduate School of Medicine and Dental Sciences, Kagoshima University
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KAMEYAMA Masaki
Department of Physiology, Graduate School of Medicine and Dental Sciences, Kagoshima University
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Kameyama Masaki
Department Of Aeronautics And Space Engineering Tohoku University
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Han Dong-Yun
School of Pharmaceutical Science, China Medical University, China
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Guo Feng
School of Pharmaceutical Science, China Medical University, China
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Hao Li-Ying
School of Pharmaceutical Science, China Medical University, China
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Minobe Etsuko
Department Of Physiology Graduate School Of Medical And Dental Sciences Kagoshima University
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Han Dong-yun
School Of Pharmaceutical Science China Medical University
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Wang Wu-yang
Department Of Physiology Graduate School Of Medical And Dental Sciences Kagoshima University
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Hao Li-ying
School Of Pharmaceutical Science China Medical University
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Xu Jian-jun
Department Of Physiology Graduate School Of Medical And Dental Sciences Kagoshima University
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Guo Feng
School Of Pharmaceutical Science China Medical University
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Xu Jian-jun
Department Of Physiology Graduate School Of Medicine And Dental Sciences Kagoshima University
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Hao Li-ying
Department Of Physiology Graduate School Of Medical And Dental Sciences Kagoshima University
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Kameyama Masaki
Department Of Physiology Graduate School Of Medical And Dental Sciences Kagoshima University
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Minobe Etsuko
Department of Physiology, Graduate School of Medical and Dental Sciences, Kagoshima University, Japan
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Xu Jian-Jun
Department of Physiology, Graduate School of Medical and Dental Sciences, Kagoshima University, Japan
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Kameyama Masaki
Department of Physiology, Graduate School of Medical and Dental Sciences, Kagoshima University, Japan
著作論文
- Calmodulin- and Ca2+-Dependent Facilitation and Inactivation of the CaV1.2 Ca2+ Channels in Guinea-Pig Ventricular Myocytes
- Calmodulin- and Ca^-Dependent Facilitation and Inactivation of the Ca_v1.2 Ca^ Channels in Guinea-Pig Ventricular Myocytes