Honma Yoshio | Department Of Life Science Shimane University Faculty Of Medicine
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概要
関連著者
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Honma Yoshio
Department Of Life Science Shimane University Faculty Of Medicine
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OKABE KADO
Research Institute for Clinical Oncology, Saitama Cancer Center
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Okabe Kado
Research Institute For Clinical Oncology Saitama Cancer Center
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Kasukabe Takashi
Research Institute For Clinical Oncology Saitama Cancer Center
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Komada Y
Mie University
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KOMADA Yoshihiro
Mie University
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Kaneko Yasuhiko
Research Institute For Clinical Oncology Saitama Cancer Center
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Komada Yoshihiro
Pediatrics And Developmental Science Institute Of Molecular And Experimental Medicine Mie University
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KASUKABE Takashi
Research Institute for Clinical Oncology, Saitama Cancer Center
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Honma Yoshio
Department Of Chemotherapy Saitama Cancer Center Research Institute
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HONMA Yoshio
Department of Chemotherapy, Saitama Cancer Center Research Institute
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Kobayashi Hirofumi
Hematology Clinic Saitama Cancer Center Hospital
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Maseki Nobuo
Hematology Clinic Saitama Cancer Center Hospital
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Honma Yoshio
School Of Medicine Shimane University
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Hanada Ryoji
Saitama Children's Medical Center
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HONMA Yoshio
Research Institute for Clinical Oncology, Saitama Cancer Center
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NAKAGAWARA Akira
Department of Biochemistry, Chiba Cancer Center Research Institute
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Nakagawara Akira
Department Of Biochemistry Chiba Cancer Center Research Institute
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AKIMOTO Miho
Department of Life Science, Shimane University Faculty of Medicine
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Akimoto Miho
Department Of Life Science Shimane University Faculty Of Medicine
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HONMA Yoshio
Department of Life Science, Shimane University Faculty of Medicine
著作論文
- Extracellular NM23 protein promotes the growth and survival of primary cultured human acute myelogenous leukemia cells
- Extracellular NM23-H1 protein inhibits the survival of primary cultured normal human peripheral blood mononuclear cells and activates the cytokine production
- Clinical significance of serum NM23-H1 protein in neuroblastoma
- Cotylenin A, a new differentiation inducer, and rapamycin cooperatively inhibit growth of cancer cells through induction of cyclin G2
- Therapeutic strategy using phenotypic modulation of cancer cells by differentiation-inducing agents