Arai Naoko | Department Of Cell Signaling Dnax Research Institute Of Molecular And Cellular Biology
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概要
- ARAI Naokoの詳細を見る
- 同名の論文著者
- Department Of Cell Signaling Dnax Research Institute Of Molecular And Cellular Biologyの論文著者
関連著者
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Arai Naoko
Department Of Cell Signaling Dnax Research Institute Of Molecular And Cellular Biology
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YOKOTA Takashi
Department, of Information Science, Graduate School of Engineering, Utsunomiya University
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ARAI Ken-ichi
Department of Chemistry and Chemical Biology, Graduate School of Engineering, Gunma University
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TAKEMOTO Naofumi
Department of Stem Cell Regulation, The Institute of Medical Science, The University of Tokyo
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ARAI Naoko
Department of Cell Signaling, DNAX Research Institute of Molecular and Cellular Biology
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Yokota Takashi
Department Of Stem Cell Regulation. The Institute Of Medical Science The University Of Tokyo
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Yokota Takashi
Department Of Analytical Chemistry Faculty Of Pharmaceutical Sciences Hoshi University
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Arai Ken-ichi
Department Of Molecular And Developmental Biology The Institute Of Medical Science The University Of
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Arai Ken-ichi
Department Of Chemistry And Chemical Biology Graduate School Of Engineering Gunma University
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Takemoto Naofumi
Department Of Molecular And Developmental Biology The Institute Of Medical Science The University Of
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Chen Wei
Department of Obstetrics and Gynecology, Kobe University Graduate School of Medicine
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Koyano‐nakagawa N
Univ. Tokyo Tokyo Jpn
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KOYANO-NAKAGAWA Naoko
Depattment of Molecular and Developmental Biology, The Imstitute of Medical Science, The University
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ARAI Ken-ichi
Depattment of Molecular and Developmental Biology, The Imstitute of Medical Science, The University
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Doyano-nakagawa Naoko
Department Of Molecular And Developmental Biology The Institute Of Medical Science The University Of
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Arai Naoko
Department Of Immunobiology Ginkgo Biomedical Research Institute
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MIYATAKE Shoichiro
Department of Molecular and Developmental Biology The Institute of Medical Science, The University o
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ISHIDA Koji
Department of Chemistry, School of Science and Engineering, Waseda University
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Ohkawa Jun
Department Of Immunobiology Ginkgo Biomedical Research Institute
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CHO Minkwon
Department of Immunobiology, Ginkgo Biomedical Research Institute
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CHEN Jingtao
Department of Immunobiology, Ginkgo Biomedical Research Institute
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NAMIKI Sahori
Department of Immunobiology, Ginkgo Biomedical Research Institute
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KOTAKI Ayumi
Department of Immunobiology, Ginkgo Biomedical Research Institute
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KAMOGAWA SCHIFTER
Department of Immunobiology, Ginkgo Biomedical Research Institute
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Cho Minkwon
Department Of Immunobiology Ginkgo Biomedical Research Institute
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Chen Jingtao
Department Of Immunobiology Ginkgo Biomedical Research Institute
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Kotaki Ayumi
Department Of Immunobiology Ginkgo Biomedical Research Institute
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Namiki Sahori
Department Of Immunobiology Ginkgo Biomedical Research Institute
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Miyatake S
Department Of Molecular And Developmental Biology The Institute Of Medical Science The University Of
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Kamogawa Schifter
Department Of Immunobiology Ginkgo Biomedical Research Institute
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Arai Ken-ichi
Department Of Immunobiology Ginkgo Biomedical Research Institute
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Chen Wei
Department Of Obstetrics And Gynecology Kobe University Graduate School Of Medicine
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Ishida Koji
Department Of Applied Photonics System Technology Faculty Of Photonics Science And Technology Chitos
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Ishida Koji
Department Of Immunobiology Ginkgo Biomedical Research Institute
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Chen Wei
Department Of Nutrition And Food Hygiene Public Health School Harbin Medical University
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Chen Wei
Department Of Food And Nutrition Providence University
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Chen Wei
Department Of Chemistry Mcgill University:(present Address)institute Of Biomedical Engineering Unive
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Chen Wei
Department Of Pediatrics Division Of Hematology Oncology And Bone Marrow Transplantation University
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Chen Wei
Department Of Chemical Engineering Nagoya University
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Chen Wei
Department of Cell Biology, Third Military Medical University
著作論文
- SAGE library screening reveals ILT7 as a specific plasmacytoid dendritic cell marker that regulates type I IFN production
- Four P-like elements are required for optimal transcription of the mouse IL-4 gene: involvement of a distinct set of nuclear factor of activated T cells and activator protein-1 family proteins
- T_h2-specific DNase I-hypersensitive sites in the murine IL-13 and IL-4 intergenic region