FUJINO Masahiro | Department of Cardiology, Fukuoka University School of Medicine

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概要

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著作論文

Purulent pericarditis with Salmonella enteritidis in a patient with CD4/CD8 depression
CXCR3 Chemokine Receptor-Plasma IP10 Interaction in Patients With Coronary Artery Disease
Nifedipine-Induced Vascular Endothelial Growth Factor Secretion from Coronary Smooth Muscle Cells Promotes Endothelial Tube Formation via the Kinase Insert Domain-Containing Receptor/Fetal Liver Kinase-1/NO Pathway
PJ-388 Cholesteryl ester transfer protein inhibitor induces antiproliferation through a Ras-independent cell-cycle regulatory pathway(Lipid Disorders 2 (IHD) : PJ65)(Poster Session (Japanese))
OJ-268 Nifedipine indirectly induces endothelial tube formation through KDR/Flk-1 by increasing the production of endothelial growth factor from smooth muscle cells(Angiogenesis 2 (M) : OJ31)(Oral Presentation (Japanese))
OJ-267 Ras/Raf/ERK-dependent pathway mediated by PTX-sensitive G protein-coupled receptors from sphingosine-1-phosphate induces human coronary artery endothelial tube formation(Angiogenesis 2 (M) : OJ31)(Oral Presentation (Japanese))
OJ-066 Revaluation of minimally or extensively oxidized low-density lipoproteins-induced extracellular signal-activated kinase 1/2 activation through Lox-1 in coronary smooth muscle cells(Lipid Disorders 1 (H) : OJ8)(Oral Presentation (Japanese))
OE-311 Constitutively active homo-oligomerization of angiotensin II type 2 receptor induces cytoplasmic cell signals independent of angiotensin II stimulation(Hypertension, Basic 3 (H) : OE39)(Oral Presentation (English))
Ox-LDL Activates P42/44 MAP Kinase via Homodimerization of LOX-1 in the Cell Membrane
Upregulation of Monocyte Surface CD11a/CD18 in Patients with Ischemic Heart Disease
A small difference in the molecular structure of angiotensin II receptor blockers induces AT_1 receptor-dependent and -independent beneficial effects
High Density Lipoprotein-lnduced Angiogenesis Requires the Activation of Ras/MAP Kinase in Human Coronary Artery Endothelial Cells

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