Nieminen P. | Department Of Pedodontics And Orthodontics Institute Of Dentistry
スポンサーリンク
概要
関連著者
-
Nieminen P.
Department Of Pedodontics And Orthodontics Institute Of Dentistry
-
Pirinen S.
Department Of Pedodontics And Orthodontics Institute Of Dentistry
-
Thesleff I.
Institute Of Biotechnology University Of Helsinki
-
ARTE S.
Department of Pedodontics and Orthodontics, Institute of Dentistry
-
PIRINEN S.
Department of Pedodontics and Orthodontics, Institute of Dentistry
-
Arte S.
Department Of Pedodontics And Orthodontics Institute Of Dentistry
-
Nieminen P.
Institute Of Dentistry And Institute Of Biotechnology University Of Helsinki
-
Peltonen L.
Department Of Human Molecular Genetics National Public Health Institute
-
Knuutila S.
Department Of Medical Genetics Haartman Institute Helsinki University Central Hospital University Of
-
KOTILAINEN J.
Institute of Dentistry, Biomedicum, PO Box 63, FIN-00014 University of Helsinki
-
AALTO Y.
Department of Medical Genetics, Haartman Institute, Helsinki University Central Hospital, University
-
PIRINEN S.
Institute of Dentistry, Biomedicum, PO Box 63, FIN-00014 University of Helsinki
-
APAJALAHTI S.
Department of Pedodontics and Orthodontics, Institute of Dentistry
-
HAAVIKKO K.
Department of Pedodontics and Orthodontics, Institute of Dentistry
-
THESLEFF I.
Department of Pedodontics and Orthodontics, Institute of Dentistry
-
Aalto Y.
Department Of Medical Genetics Haartman Institute Helsinki University Central Hospital University Of
-
Aalto Y.
Department Of Medical Genetics Haartman Institute And Helsinki University Central Hospital Universit
-
Kotilainen J.
Institute Of Dentistry Biomedicum Po Box 63 Fin-00014 University Of Helsinki
-
Apajalahti S.
Department Of Pedodontics And Orthodontics Institute Of Dentistry
-
Haavikko K.
Department Of Pedodontics And Orthodontics Institute Of Dentistry
著作論文
- MSX1 Gene is Deleted in Wolf-Hirschhorn Syndrome Patients with Oligodontia
- Characteristics of Incisor-Premolar Hypodontia in Families
- Gene Defect in Hypodontia : Exclusion of EGF, EGFR, and FGF-3 as Candidate Genes