Tuji Keiko | Department Of Cardiology Kyoto University School Of Medicine

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著作論文

Hydroxyzine, a First Generation H_1-Receptor Antagonist, Inhibits Human Ether-a-go-go-Related Gene (HERG) Current and Causes Syncope in a Patient With the HERG Mutation
PJ-571 Multiple Mechanisms underlie Long QT Syndrome by the Change of Amino Acid R259 in the Intracellular S4-S5 Linker of KCNQ1(PJ096,Arrhythmia, Others (Clinical/Pathophysiology) 4 (A),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The
PE-106 SCN5A and Lamin A/C Gene Mutations are Highly Prevalent in Patients with Familial Bradyarrhythmic Disorders(PE018,Arrhythmia, Others (Clinical/Pathophysiology) (A),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulat
A novel SCN5A gain-of-function mutation M1875T associated with familial atrial fibrillation
LQT1, but not LQT2, patients show an exercise-induced increase in transmural dispersion of repolarization (TDR)
FRS-052 Identification and Functional Characterization of a Novel Intronic Splicing Mutation in KCNQ1 Associated with Long QT Syndrome(FRS11,New Insights into Basic Mechanisms of Arrhythmia (A),Featured Research Session (English),The 73rd Annual Scientifi
Mutation Analysis of the Glycerol-3 Phosphate Dehydrogenase-1 Like (GPD1L) Gene in Japanese Patients With Brugada Syndrome
Mutation Site Dependent Variability of Cardiac Events in Japanese LQT2 Form of Congenital Long-QT Syndrome
A Novel Mutation Associated With Jervell and Lange-Nielsen Syndrome in a Japanese Family
PE-251 Mutation analysis of the Glycerol-3 Phosphate Dehydrogenase-1 Like (GPD1L) Gene in Japanese Patients with Brugada Syndrome(Arrhythmia, diagnosis/Pathophysiology/EPS(07)(A),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese C
OE-391 Cardiac sodium channel gene mutations are prevalent in Japanese patients with familial sick sinus syndrome(Arrhythmia, diagnosis/Pathophysiology/EPS(02)(A),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation So
FRS-109 A novel SCN5A gain-of-function mutation M1875T associated with familial atrial fibrillation(New Horizon in Diagnosis and Pathophysiology of Arrhythmia(A),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Soci
3 Mutant KCNE3 Reduces Repolarizing Potassium Current And Causes Long QT Syndrome(Plenary Session 4 (PL-4) (A) New Scientific Approaches to QT Prolongation,Special Program,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OJ-009 N- And C-terminal KCNE1 Mutations Cause Distinct Phenotypes of Long QT Syndrome(Arrhythmia, diagnosis/pathophysiology/EPS-03, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
1 Brugada Syndrome and Diverse Phenotypes of Cardiac Sodium Channelopathies(Symposium 5 (SY-05) (A) New Findings of Mechanism and Therapeutic Strategies of Idiopathic Ventricular Fibrillation,Special Program,The 72nd Annual Scientific Meeting of the Japan
PJ-046 Genotype-Phenotype Correlations of KCNJ2 Mutations in Japanese Patients with Andersen-Tawil Syndrome(Arrhythmia, diagnosis/pathophysiology/EPS-10, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OJ-007 Variable Genetic Backgrounds Between Congenital and Acquired Long-QT Syndromes(Arrhythmia, diagnosis/pathophysiology/EPS-03, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-081 Mechanistic Basis for the Pathogenesis of Long QT Syndrome Associated with a Common Splicing Mutation in KCNQ1 Gene(Arrhythmia, basic-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
FRS-078 Spectrum and Prevalence of Cardiac Sodium Channel (SCN5A) Mutations in a Cohort of 194 Unrelated Patients Referred for Inherited Arrhythmias(Arrhythmia (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OJ-349 High Incidence of Cardiac Events for Long-QT Syndrome with Compound Mutations(Arrhythmia, diagnosis/pathophysiology/EPS-4 (A) OJ59,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Genetic Analysis of PRKAG2 in a Family with Inherited Wolff-Parkinson-White Syndrome (Arrhythmia, Diagnosis/Pathophysiology/EPS 8 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
High-risk for Bradyarrhythmic Complications in Patients with Brugada Syndrome Caused by SCN5A Gene Mutations(Arrhythmia, Basic (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Quantitative Analysis of Aberrant Splicing Caused by a Splice-site Mutation in KCNQ1 Gene of a Patient with Long QT Syndrome (Genetics/Genetically Engineered Models (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
SCN5A Gene Silencing by Adenovirus-mediated Short Interfering RNA Delivery in Neonatal Rat Ventricular Myocytes(Arrhythmia, Basic 2 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)

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