Murasawa Satoshi | Department Of Medicine Ii Kansai Mesical University
スポンサーリンク
概要
関連著者
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Murasawa Satoshi
Department Of Medicine Ii Kansai Mesical University
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Murasawa Satoshi
Department of Medicine II, Kansai Medical University
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Murasawa Satoshi
Department Of Regenerative Medicine Institute Of Biomedical Research And Innovation Of Kobe
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Mori Yasukiyo
Department of Cardiovascular Medicine, Kyoto Prefectural University School of Medicine
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Iwasaka Toshiji
Department of Medicine 2 Kansai Medical University
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MARUYAMA Katsuya
Department of Internal Medicine, Kurihama National Hospital
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Maruyama Katsuya
The Department Of Medicine Ii Kansai Medical University
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Mori Yasukiyo
Department Of Cardiology Kyoto Prefectural University Of Medicine
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Iwasaka Toshiji
Department Of Internal Medicine 2 Kansai Medical University
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Miyasaka Yoko
Second Department Of Internal Medicine Division Of Cardiology Kansai Medical University
著作論文
- IS005 Vascular Smooth Muscle Cell(VSMC)-Specific Overexpression of Angiotensin type2(AT2)Receptor Causes Endothelium-dependent Vasodilation via bradykinin/NO/cGMP pathway
- IS104 Cell-specific Distribution of Angiotensin II Receptor Subtypes and Receptors-mediated Biochemical Changes in Failing Human Hearts
- IS040 Angiotensin Type 2 (AT2) Receptor Inhibits AT1-mediated Intracelluar Alkalosis to Enhance Kinik Production in Vascular Smooth Muscle Cells From Vascular-targeted AT2 Transgenic Mice
- IS070 Angiotensin Type 2 Receptors Are Re-expressed by Cardiac Fibroblasts from Failing Myopathic Hamster Hearts and Inhibit Cell Growth and Fibrillar Collagen Metabolism
- IS050 Cardiac-Specific Overexpression of Angiotensin AT2 receptor Causes Attenuated Response to AT1 receptor - Mediated Chronotropic Action
- Down-Regulation by cAMP of Angiotensin II Type 2 Receptor Gene Expression in PC12 Cells
- RAT ANGIOTENSIN II(TYPE 1A)RECETOR _mRNA REGULATION AND ITS SUBTYPES EXPRESSION IN THE MYOCARDIAL GROWTH AND HYPERTROPHY
- Phase I/II Clinical Trial : Transplantation of Autologous Endothelial Progenitor Cells in Patients with Chronic Critical Limb Ischemia(Angiogenesis 1 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)