Nakaoka Yoshikazu | Department of Structural Analysis, National Cardiovascular Center, Research Institute

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著作論文

OE-316 Gab Family Scaffolding Adaptor Proteins are Essential Mediators in the Signal Circuit between Myocardium and Endothelium(Molecular biology, myocardium-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-132 Gab Family Proteins Are Essential for the Prevention of Heart Failure(Heart failure, basic-1 (M) OE22,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Clinical significance of plasma endothelin-1 level after bosentan administration in pulmonary arterial hypertension
FRS-003 Docking Protein Gab1 is Essential for HGF-dependent Endothelial Signaling and Angiogenesis after Hindlimb Ischemia(FRS1,New Molecules for Cardiovascular Regulation (M),Featured Research Session (English),The 73rd Annual Scientific Meeting of The J
OE-172 Gab2 is Essential for Angiopoietin-1/Tie2-dependent Signaling and Cytoprotection in Endothelial Cells(Molecular biology, vascular(01)(IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PJ-761 Gab1-SHP2-ERK Signaling Pathway Comprises and Inhibitory Axis for Insulin-like Growth Factor-1-induced Myogenic Differntiation(Development and differentiation-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-113 Clinical Significance of Plasma Endothelin-1 Level after Bosentan Administration in Pulmonary Arterial Hypertension(OE19,Pulmonary Circulation (H),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
FRS-024 Interaction of Scaffolding/Docking Protein Gab1 with Tyrosine Phosphatase SHP2 Is Involved in gp130-dependent Activation of ERK5 and Elongation of Cardiomyocytes(Cardiac Hypertrophy (M) : FRS3)(Featured Research Session (English))
Bcl-xL Is Up-Regulated in the Hearts of Cardiac-Specific Smad1 Transgenic Mice
Targeted Disruption of Gab1 Gene Leads to Developmental Abnormalities in Heart
Cardiac-specific activation of STAT3 promotes the vessel growth in the hearts
Specific Cardiomyopathy Caused by Multisystemic Lipid Storage in Jordans' Anomaly
OJ-523 Skeletal Alpha-actin Gene Expression Is Negatively Regulated by Protein Tyrosine Phosphatase SHP2 Through Inhibition of Rho signaling Pathway in Cardiomyocytes(Molecular Biology, Myocardium 2 (M) : OJ64)(Oral Presentation (Japanese))
OJ-436 Mutations of bone morphogenetic protein receptor 2 gene and polymorphism of serotonin transporter gene in patients with primary pulmonary hypertension(Pulmonary Circulation 1 (H) : OJ53)(Oral Presentation (Japanese))
OE-294 Overexpression of Smad1 Protects against Myocardial Ischemia-reperfusion Injury through Induction of Bcl-xL in Transgenic Mice(Genetics/Genetically Engineered Models 2 (H) : OE36)(Oral Presentation (English))
Bone Morphogenetic Protein-2 Signaling through Smad1 Inhibits Pulmonary Artery Smooth Muscle Cell Proliferation
Stat3 Activation Is Responsible for Protecting Cardiac Fibrosis through Inhibiting Stretch-Induced TGFbeta : Generation and Characterization of Cardiac-Specific StatS-Deficient Mice
PE-472 AMP-activated Protein Kinase Regulates Protein Translation through Phosphorylation of eEF2 During Hypoxia in Cardiomyocyte(Myocardial Ischemia-Reperfusion, Basic (M) : PE80)(Poster Session (English))
1 The Novel Downstream Target of Neuregulin-1/ErbB Signaling Pathway for Maintenance of Cardiac Function(Interaction between Myocardium and Vasculature in Heart Failure-Novel Insights from Cytokine Signal Network-,Symposium 8 (SY-08) (IHD),The 73rd Annual

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