Li Yiwen | Department of Cardiology, Respirology and Nephrology, Gifu University Graduate School of Medicine

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著作論文

PJ-225 Tropomyosin as One of the Earliest Target of Apoptotic Proteolysis Facilitating Cytoskeletal Catastrophe in Cardiac Myocytes(Heart Failure, Basic 2 (M) : PJ38)(Poster Session (Japanese))
PE-385 G-CSF Upregulates Myocardial Akt and Improves Function of the Hearts with Established Heart Failure due to Large Old Myocardial Infarction(Old Myocardial Infarction/Remodeling 3 (IHD) : PE66)(Poster Session (English))
OJ-508 Autophagy, but Not Apoptosis, May be the Mode of Myocyte Death in Hearts of the Cardiomyopathic UM-X7.1 Strain Hamster(Cardiomyopathy, Basic (M) : OJ62)(Oral Presentation (Japanese))
PE-304 Molecular Mechanisms for Beneficial Effects of Fas Signal Inhibition by the Soluble Fas Gene Therapy on Doxorubicin-induced Cardiomyopathy(Cardiomyopathy, basic/clinical-4, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PJ-404 Angiotensin II Type 1 Receptor Blocker improves Postinfarction Ventricular Remodeling by Inhibition of Granulation Tissue Cell Apoptosis(Heart failure, basic-5 (M) PJ68,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the
PE-388 Reduction of Inflammatory Cytokine Expression and Oxidative Damage by Erythropoietin and Its Molecular Signals in Chronic Heart Failure(Heart failure, basic-4 (M) PE65,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the J
OE-131 G-CSF Displays Its Beneficial Effects on Postinfarction Cardiac Function by Cardiomyocyte Hypertrophy and Anti-fibrosis, but Not by Angiogenesis, Regeneration(Heart failure, basic-1 (M) OE22,Oral Presentation (English),The 70th Anniversary Annual S
Hepatocyte Growth Factor Gene Therapy Improves Cardiac Function of Doxorubicin-induced Cardiomyopathy(Gene Therapy 3 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Erythropoietin Improves Cardiac Function of Doxorubicin-induced Cardiomyopathy(Cardiac Hypertrophy/Cardiomyopathy 1 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Soluble Fas Gene Therapy Improves Cardiac Function of Doxorubicin-induced Cardiomyopathy, Independently of Cardiomyocyte Apoptosis(Cardiomyopathy, Basic (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OE-264 Autophagic Cardiomyocyte Death in Cardiomyopathic Hamsters and Its Prevention by Granulocyte Colony-Stimulating Factor(Cardiomyopathy, basic/clinical-1 (M) OE45,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japan
PE-428 Cyclophosphamide Improves Left Ventricular Dysfunction in Post-MI Chronic Heart Failure by Downregulating Inflammatory Cytokine and MMP(Heart failure, basic-3, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-082 5-Fluorouracil Improves Postinfarct Left Ventricular Dysfunction due to Upregulated SDF-1, Mobilized CD34 Positive Cells and Attenuated Fibrosis(Acute myocardial infarction, basic-1 (IHD) OE14,Oral Presentation (English),The 70th Anniversary Annual
PE-386 Combination Thearpy of Antiapoptosis and Regeneration against Postinfarct Heart Failure : A Novel Therapeutic Concept(Old Myocardial Infarction/Remodeling 3 (IHD) : PE66)(Poster Session (English))
OE-324 Postinfarction Gene Transfer of Soluble TGF-β Typed Receptor Relieves Heart Failure during the Chronic Stage of Myocardial Infarction(Gene Therapy 2 (IHD) : OE41)(Oral Presentation (English))
Gene Transfer of Soluble TGF-β Type II Receptor Suppresses Cardiac Fibrosis and Improves Cardiac Function in Cardiomyopathic Hamsters
Nerve growth factor and the neurotrophic factor hypothesis
Myocardial Apoptosis Index Based on DNA in Situ Nick End-Labeling (TUNEL) Can not Predict Prognosis of Patients with Dilated Cardiomyopathy
Myocytes Positive for in Situ Markers for DNA Breaks in Hypertrophic, but Neither Failed Nor Dilated, Human Hearts
Granulocyte-Colony Stimulating Factor (G-CSF) Prevents Myocardial Fibrosis and Heart Failure in Cardiomyopathic Hamsters
Fluctuation of Intracellular Ca^ through Multiple Ca^ Regulatory Proteins May Be Inevitable for Initiating Apoptosls in Adult Cardiac Myocytes

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