スポンサーリンク
日本酸化ストレス学会 | 論文
- Suppressive Effect of Curcumin on Lipid Peroxidation Induced in Rats by Carbon Tetrachloride or 60Co-Irradiation.
- Plasma copper and zinc and their relationships with plasma lipids, lipoproteins, lipase activity, and platelet aggregation in hypertension and diabetes.
- Plasma Lipoprotein(a) Levels Showing Age-Related Changes in Diabetic and Hypertensive Individuals.
- Changes in Body Iron Status, Heart Rate, and Unspecific Symptoms across Menstrual Cycle in Marginally Iron-Deficient Young Women.
- Increased Intestinal Folate Absorption in Experimental Diabetes.
- Metabolism and Urinary Excretion of Folates in Hypothyroid Rats.
- Increased Sensitivity of Platelets from Unstable Angina Patients to Adenosine Diphosphate-Induced Aggregation.
- Relationships among Plasma Lipids, Lipoproteins, and Platelet Aggregation Profiles in Essential Hypertension
- Glycosylation and Placental Transport of Immunoglobulin G.
- Lipid Peroxidation of Microsomal Membranes as the Mechanism of 4-Aminopyrazolopyrimidine-Induced Cell Injury in the Rat Liver.
- Vitamin D Improves Oral Glucose Tolerance and Insulin Secretion in Human Diabetes.
- Anterior Segment Collagenase Activity in Children with Keratomalacia.
- Inhibition of Oxygen-induced Retinal Neovascularization in the Neonatal Rat by Green Tea Extract
- Elevation of the Mitochondrial Oxidative Activity of Rat Liver by Long-Term Intake of a High-Fat Diet Based on Beef Tallow or Safflower Oil.
- Protective Role of .ALPHA.-Tocopherol Acetate against Thioacetamide-Induced Mitochondrial Dysfunction.
- Electrical Stimulation of the Lateral Part of the Dorsal Parabrachial Nucleus Causes Hyperglycemia
- Detection of Enhanced Lipid Peroxide Levels in Patients with Inflammatory Skin Diseases
- Serum Amino Acid Pattern of Type II Citrullinemic Patients and Effect of Oral Administration of Citrulline
- Involvement of Activated Polymorphonuclear Leukocytes in Galactosamine-Induced Hepatic Injury in Rats.
- Protective Effect of Phyllanthus fraternus against Thioacetamide-Induced Mitochondrial Dysfunction.