根未完成歯の不完全脱臼による予後に関する病理組織学的研究

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Experimental incomplete luxations were undertaken in immature rat molars to understand the reactions of periodontal and pulp tissues. The rats were sacrificed immediately after the procedure and after 1 day, 3 days, 7 days, 14 days, 30 days or 60 days to compare histopathological findings in the experimental group with those in the control group, inaddition to the extracted teeth (complete luxation). The following results were obtained. 1. Immediately after surgery, fagmentation hemorrhage, and regressive changes were observed in the periodontal tissues, and degeneration and disappearance were seen especially at the epithelium of the root apical region in the early stage after surgery. 2. Repair of the damaged periodontal tissues was seen 7 and 14 days after surgery, but irregular thickening and proliferation of cellular cementum at the apical region were found. 3. Thirty days postoperatively, the irregularity of the root shape and anomaliesof root formation at the apical regions were observed. It appears that the irregularity of root formation were related to the degeneration and disapperance of the epithelial sheath due to incomplete luxation, as well as to the effect of non-splinting. 4. Immediately after incomplete luxation, the characteristic ablations of the odontoblast layer of dentin were seen with retention of a fluid component in the ablated spaces. One and 3 days after surgery, cellular components suchas erythrocytes and leukocytes were seen. 5. Ablations of the odontoblast layer were seen in the coronal pulp and those occurred most frequently in the tectorium of the pulp horn, cervical area and floor of the pulp chamber. Similar findings were observed in the extracted teeth. 6. Seven days after surgery, ablation of the odontoblast layer disappeared and inclusion of the cellular components, as well as formation of irregular dentin with small excavations and calcification were seen, and those appeared in coincidence with the ablated regions. 7. Between 7 and 60 days postoperatively, the pulpal cavities were constricted by gradual thickening and proliferation due to irregular dentin and calcification. It is suggested that the ablations of the odontblast layer of dentin after incomplete luxation in immature teeth induced formation of irregular dentin and calcification and this may be one of the reasons for pulpal obliteration after luxation injuries.
福岡歯科大学学会の論文
1994-12-31

根未完成歯の不完全脱臼による予後に関する病理組織学的研究

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