スポンサーリンク
Particle Radiation Oncology Research Center Research Reactor Institute Kyoto University | 論文
- 242 Analysis of apoptosis induction in Nbs1 deficient cells(Apoptosis related, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
- 206 Functional Domain Analysis of NBS1 Gene in Homologous Recombinational Repair(Physics, chemistry and DNA damage, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
- 98 Localization of Rad51 protein in cells from patients with Fanconi Anemia D1 group(Repair of radiation damage, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
- S-3-1 Mechanisms of DNA double-strand break repair(Biological Effects of Low Dose and Low Dose-rate Irradiations, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
- Mutation screening for a novel cancer-predisposition syndrome of total PCS
- p53-Binding Protein 1 (53BP1) is Involved in the Repair of DNA Double-Strand Breaks
- Analysis of the function of NBS1 in DNA double-strand break repair
- Histone H2AX regulates the formation of NBS1 foci on DNA damage sites
- Dissociation of ATM/NBS1 functions in telomere maintenance
- NBS1 is involved in recognition of DNA damage and initiation of homologous recombination.
- Bystander mutagenic effect of alpha particle in human-hamster hybrid cells
- Somatic Mutation Caused by Low Dose Rate Tritium Radiation : Studies Using A Hyper-sensitive DetectionSystem
- Complementation studies of immortalized cell lines from infants with total PCS
- Establishement of mouse Nbs1-deficient cell lines by gene targeting
- Association of cells from Fanconi anemia patients with radiation sensitivity
- Estimation of Dose Absorbed Fraction for ^I-bera rays in Rat Thyroid
- Association of DNA repair protein NBS1 with telomere maintenance
- The function of NBS1 and histone H2AX in the early process of DNA repair.
- Reversed dose-rate effect of high LET radiation in mutation induction
- Chromosomal instability syndrome of total premature chromatid separation with mosaic variegated aneuploidy is defective in mitotic spindle checkpoint